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Merck
CN

139181

Bromodichloromethane

≥97%

Synonym(s):

Dichlorobromomethane

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About This Item

Linear Formula:
BrCHCl2
CAS Number:
75-27-4
Molecular Weight:
163.83
Beilstein:
1697005
EC Number:
200-856-7
MDL number:
MFCD00000824
UNSPSC Code:
12352100
PubChem Substance ID:
24848395
NACRES:
NA.22

Key Documents

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Quality Level

100

Assay

≥97%

form

liquid

contains

potassium carbonate as stabilizer

refractive index

n20/D 1.497 (lit.)

bp

87 °C (lit.)

mp

−55 °C (lit.)

solubility

water: insoluble

density

1.98 g/mL at 25 °C (lit.)

functional group

bromo
chloro

SMILES string

ClC(Cl)Br

InChI

1S/CHBrCl2/c2-1(3)4/h1H

InChI key

FMWLUWPQPKEARP-UHFFFAOYSA-N

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Application

Bromodichloromethane was used to investigate the in vivo genotoxicity of chlorodibromomethane, bromodichloromethane and bromoform in mice.

Biochem/physiol Actions

Bromodichloromethane (BDCM) induces steatohepatitic injury in human liver. BDCM induces the cell proliferation in different tissues and kidney damage by renal tuble cell proliferation in male F344 rats.

Signal Word

Danger

Hazard Statements

H302,H315,H318,H335,H351

Hazard Classifications

Acute Tox. 4 Oral - Carc. 2 - Eye Dam. 1 - Skin Irrit. 2 - STOT SE 3

Target Organs

Respiratory system

Storage Class Code

10 - Combustible liquids

WGK

WGK 3

Flash Point(F)

Not applicable

Flash Point(C)

Not applicable

Personal Protective Equipment

dust mask type N95 (US), Eyeshields, Gloves

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Certificates of Analysis (COA)

Lot/Batch Number
MKCZ8417
MKCZ3657
MKCX8242
MKCX5571
MKCR7815

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K J Stocker et al.
Mutagenesis, 12(3), 169-173 (1997-05-01)
Chlorination of drinking water results in the formation of chlorodibromomethane, bromodichloromethane and bromoform. These trihalomethanes have all shown evidence of genotoxicity in bacterial and mammalian cell systems in vitro and some evidence of carcinogenicity in rodents. Chlorodibromomethane and bromodichloromethane have
Richard J Summerhayes et al.
Epidemiology (Cambridge, Mass.), 23(1), 15-22 (2011-12-14)
Trihalomethanes in drinking water have been associated with higher occurrence of small-for-gestational-age (SGA) births, although results have been inconsistent. We geocoded residential address for mother of live, singleton, term births to 33 water distribution systems in a large metropolitan area
Ratanesh Kumar Seth et al.
Toxicology and applied pharmacology, 274(1), 42-54 (2013-11-12)
Environmental toxins induce a novel CYP2E1/leptin signaling axis in liver. This in turn activates a poorly characterized innate immune response that contributes to nonalcoholic steatohepatitis (NASH) progression. To identify the relevant subsets of T-lymphocytes in CYP2E1-dependent, environment-linked NASH, we utilized
Suvarthi Das et al.
American journal of physiology. Gastrointestinal and liver physiology, 305(12), G950-G963 (2013-10-26)
Recent studies indicate that metabolic oxidative stress, autophagy, and inflammation are hallmarks of nonalcoholic steatohepatitis (NASH) progression. However, the molecular mechanisms that link these important events in NASH remain unclear. In this study, we investigated the mechanistic role of purinergic
Minh T Do et al.
Environmental health perspectives, 113(4), 418-424 (2005-04-07)
Chlorination disinfection by-products (CDBPs) are produced during the treatment of water with chlorine to remove bacterial contamination. CDBPs have been associated with an increased risk of bladder cancer. There is also some evidence that they may increase the risk of
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