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About This Item
Empirical Formula (Hill Notation):
Cu
CAS Number:
Molecular Weight:
63.55
NACRES:
NA.23
PubChem Substance ID:
UNSPSC Code:
12141711
MDL number:
InChI
1S/Cu
SMILES string
[Cu]
InChI key
RYGMFSIKBFXOCR-UHFFFAOYSA-N
assay
99.95%
form
foil
manufacturer/tradename
Goodfellow 296-290-08
resistivity
1.673 μΩ-cm, 20°C
diam. × thickness
15 mm × 0.075 mm
bp
2567 °C (lit.)
mp
1083.4 °C (lit.)
density
8.94 g/mL at 25 °C (lit.)
General description
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Donita C Brady et al.
Nature, 509(7501), 492-496 (2014-04-11)
The BRAF kinase is mutated, typically Val 600→Glu (V600E), to induce an active oncogenic state in a large fraction of melanomas, thyroid cancers, hairy cell leukaemias and, to a smaller extent, a wide spectrum of other cancers. BRAF(V600E) phosphorylates and
Hiroki Serizawa et al.
Organic letters, 16(13), 3456-3459 (2014-06-14)
The direct synthesis of pentafluoroethyl copper (CuC2F5) from a cuprate reagent and ethyl pentafluoropropionate as one of the most economical and useful pentafluoroethyl sources was accomplished. The advantages of this method are; all the reagents employed are low-cost and operationally
Jing Zheng et al.
Organic letters, 16(13), 3560-3563 (2014-06-25)
A Rh(III)-catalyzed selective coupling of N-methoxy-1H-indole-1-carboxamide and aryl boronic acids is reported. The coupling is mild and efficient toward diverse product formation, with selective C-C and C-C/C-N bond formation. Kinetic isotope effects studies were conducted to reveal a mechanism of
Lelita T Braiterman et al.
Proceedings of the National Academy of Sciences of the United States of America, 111(14), E1364-E1373 (2014-04-08)
Wilson disease (WD) is a monogenic autosomal-recessive disorder of copper accumulation that leads to liver failure and/or neurological deficits. WD is caused by mutations in ATP7B, a transporter that loads Cu(I) onto newly synthesized cupro-enzymes in the trans-Golgi network (TGN)
Julie E Gleason et al.
Proceedings of the National Academy of Sciences of the United States of America, 111(16), 5866-5871 (2014-04-09)
The human fungal pathogens Candida albicans and Histoplasma capsulatum have been reported to protect against the oxidative burst of host innate immune cells using a family of extracellular proteins with similarity to Cu/Zn superoxide dismutase 1 (SOD1). We report here
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