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GF63169094

Copper - O.F.H.C.

foil, 6mm disks, thickness 0.075mm, half hard, 99.95+%

Synonym(s):

Copper, CV000540

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About This Item

Empirical Formula (Hill Notation):
Cu
CAS Number:
Molecular Weight:
63.55
NACRES:
NA.23
PubChem Substance ID:
UNSPSC Code:
12141711
MDL number:
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Product Name

Copper - O.F.H.C., foil, 6mm disks, thickness 0.075mm, half hard, 99.95+%

InChI

1S/Cu

SMILES string

[Cu]

InChI key

RYGMFSIKBFXOCR-UHFFFAOYSA-N

assay

99.95%

form

foil

manufacturer/tradename

Goodfellow 631-690-94

resistivity

1.673 μΩ-cm, 20°C

diam. × thickness

6 mm × 0.075 mm

bp

2567 °C (lit.)

mp

1083.4 °C (lit.)

density

8.94 g/mL at 25 °C (lit.)

Legal Information

Product of Goodfellow

General description

For updated SDS information please visit www.goodfellow.com.

Regulatory Information

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Donita C Brady et al.
Nature, 509(7501), 492-496 (2014-04-11)
The BRAF kinase is mutated, typically Val 600→Glu (V600E), to induce an active oncogenic state in a large fraction of melanomas, thyroid cancers, hairy cell leukaemias and, to a smaller extent, a wide spectrum of other cancers. BRAF(V600E) phosphorylates and
Hiroki Serizawa et al.
Organic letters, 16(13), 3456-3459 (2014-06-14)
The direct synthesis of pentafluoroethyl copper (CuC2F5) from a cuprate reagent and ethyl pentafluoropropionate as one of the most economical and useful pentafluoroethyl sources was accomplished. The advantages of this method are; all the reagents employed are low-cost and operationally
Giulia Cheloni et al.
Aquatic toxicology (Amsterdam, Netherlands), 155, 275-282 (2014-07-30)
The present study showed the important role of light intensity and spectral composition on Cu uptake and effects on green alga Chlamydomonas reinhardtii. High-intenisty light (HL) increased cellular Cu concentrations, but mitigated the Cu-induced decrease in chlorophyll fluorescence, oxidative stress
Lelita T Braiterman et al.
Proceedings of the National Academy of Sciences of the United States of America, 111(14), E1364-E1373 (2014-04-08)
Wilson disease (WD) is a monogenic autosomal-recessive disorder of copper accumulation that leads to liver failure and/or neurological deficits. WD is caused by mutations in ATP7B, a transporter that loads Cu(I) onto newly synthesized cupro-enzymes in the trans-Golgi network (TGN)
Adam Southon et al.
Metallomics : integrated biometal science, 5(10), 1346-1356 (2013-08-02)
Copper (Cu) is an essential redox active metal that is potentially toxic in excess. Multicellular organisms acquire Cu from the diet and must regulate uptake, storage, distribution and export of Cu at both the cellular and organismal levels. Systemic Cu

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