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EMU157701

MISSION® esiRNA

targeting mouse Id4

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About This Item

NACRES:
NA.51
UNSPSC Code:
41105324

Key Documents

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Product Name

MISSION® esiRNA, targeting mouse Id4

description

Powered by Eupheria Biotech

product line

MISSION®

form

lyophilized powder

esiRNA cDNA target sequence

TCAGCAAATCTAGAAACGTCGTCTCGTCTTGTCATTCCAAGAGAGAGAGAGAGAGAGAGAGAGAAGGGGAAAAATAAAACTTAAATTCACTTTTACTTTTTTTGCACGTTCACGAGCATTCACCGTACGTATTCTCTTTCGTTTCTTCTTTTATGACCGCTGTGAATTGTACGTTTCTGTGGTTATTTTTATCACCCTTTTGAAGGTGCAGTTAAACTTCGAAGCTTAAGTGTTGTCGACCAGACTGCTAAGTAGAAGAGCAATCGTGAATCCAACCTTAGAGGCTACATTGTGACAAGGGAACTGTTTTTGTTTTTGAAGCTTTACTAATATACCAGAGCACTGTAGATATGTTGTTTTACATCTATTGTTTAAAATAGATGATTATAACAGGGCGGGGAACTTTTTCTCTGCAAGAATGTTACATATTGTATAGATAAGTGAGTGACATTTCATACCCTGTATATATAGAGATGTTCTATAAGTGTGAGAAAGTATATGCGCTTTAAT

Ensembl | mouse accession no.

ENSMUSG00000021379

NCBI accession no.

NM_031166

shipped in

ambient

storage temp.

−20°C

Quality Level

100

Gene Information

mouse ... ID4(15904), Id4(15904)

General description

MISSION® esiRNA are endoribonuclease prepared siRNA. They are a heterogeneous mixture of siRNA that all target the same mRNA sequence. These multiple silencing triggers lead to highly-specific and effective gene silencing.

For additional details as well as to view all available esiRNA options, please visit SigmaAldrich.com/esiRNA.

Legal Information

MISSION is a registered trademark of Merck KGaA, Darmstadt, Germany

Storage Class

10 - Combustible liquids

flash_point_f

Not applicable

flash_point_c

Not applicable

Regulatory Information

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Se-Jin Park et al.
Laboratory animal research, 27(4), 333-338 (2012-01-11)
The cancer stem cell (CSC) hypothesis proposes that CSCs are the root of cancer. CSC-targeted therapies may prevent cancer relapse and provide more effective treatment. The expression of aldehyde dehydrogenase 1, as assessed by the Aldefluor assay, has been recognized
Simon Junankar et al.
Nature communications, 6, 6548-6548 (2015-03-31)
Basal-like breast cancer (BLBC) is a heterogeneous disease with poor prognosis; however, its cellular origins and aetiology are poorly understood. In this study, we show that inhibitor of differentiation 4 (ID4) is a key regulator of mammary stem cell self-renewal

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