420107
溴代三(二甲基氨基)磷鎓六氟磷酸盐
98%
别名:
BroP
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关于此项目
线性分子式:
BrP[N(CH3)2]3PF6
化学文摘社编号:
分子量:
388.07
UNSPSC Code:
12352101
PubChem Substance ID:
MDL number:
产品名称
溴代三(二甲基氨基)磷鎓六氟磷酸盐, 98%
InChI
1S/C6H18BrN3P.F6P/c1-8(2)11(7,9(3)4)10(5)6;1-7(2,3,4,5)6/h1-6H3;/q+1;-1
SMILES string
F[P-](F)(F)(F)(F)F.CN(C)[P+](Br)(N(C)C)N(C)C
InChI key
XELPBWPBGHCIKX-UHFFFAOYSA-N
assay
98%
reaction suitability
reaction type: Coupling Reactions
mp
>300 °C (lit.)
application(s)
peptide synthesis
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Application
Reagent for:
Conjugate addition of nitroalkanes to an acrylate equivalent
A novel polymer supported approach to nucleoside modification
Asymmetric hydrogenations
Peptide coupling
Pyrrolidine hydroxylation
Synthesis of cyclic PNA-based compound directed against HIV-1 TAR RNA
Conjugate addition of nitroalkanes to an acrylate equivalent
A novel polymer supported approach to nucleoside modification
Asymmetric hydrogenations
Peptide coupling
Pyrrolidine hydroxylation
Synthesis of cyclic PNA-based compound directed against HIV-1 TAR RNA
Packaging
Bottomless glass bottle. Contents are inside inserted fused cone.
signalword
Danger
hcodes
Hazard Classifications
Carc. 1B - Skin Corr. 1B
存储类别
6.1C - Combustible acute toxic Cat.3 / toxic compounds or compounds which causing chronic effects
wgk
WGK 3
flash_point_f
Not applicable
flash_point_c
Not applicable
ppe
Eyeshields, Faceshields, Gloves, type P3 (EN 143) respirator cartridges
法规信息
新产品
此项目有
V R Scott et al.
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S C Posey et al.
The Journal of biological chemistry, 274(7), 4259-4265 (1999-02-06)
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A M Senderowicz et al.
Journal of the National Cancer Institute, 87(1), 46-51 (1995-01-04)
Jasplakinolide, a cyclodepsipeptide produced by an Indo-Pacific sponge, Jaspis johnstoni, has been reported to inhibit the growth of breast cancer cells. The effects of jasplakinolide on the proliferation of three human immortalized prostate carcinoma cell lines (PC-3, LNCaP, and TSU-Pr1)
J A Rosado et al.
The Journal of biological chemistry, 275(11), 7527-7533 (2000-03-14)
The nature of the mechanism underlying store-mediated Ca(2+) entry has been investigated in human platelets through a combination of cytoskeletal modifications. Inhibition of actin polymerization by cytochalasin D or latrunculin A had a biphasic time-dependent effect on Ca(2+) entry, showing
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