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关键词:'C3868'
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Kathy Ye Morgan et al.
Tissue engineering. Part A, 20(11-12), 1654-1667 (2014-01-15)
Electrical and mechanical stimulation have both been used extensively to improve the function of cardiac engineered tissue as each of these stimuli is present in the physical environment during normal development in vivo. However, to date, there has been no
Sandra Furlan et al.
Frontiers in neuroanatomy, 14, 15-15 (2020-05-07)
Calcium stores in neurons are heterogeneous in compartmentalization and molecular composition. Danio rerio (zebrafish) is an animal model with a simply folded cerebellum similar in cellular organization to that of mammals. The aim of the study was to identify new
Calsequestrin 2 and arrhythmias
Faggioni M and Knollmann B C.
American Journal of Physiology. Heart and Circulatory Physiology, 302(6), H1250-H1260 (2011)
Absence of calsequestrin 2 causes severe forms of catecholaminergic polymorphic ventricular tachycardia
Postma A V, et al.
Circulation Research, 91(8), e21-e26 (2002)
Sandra Furlan et al.
Journal of muscle research and cell motility, 37(1-2), 27-39 (2015-11-21)
Calsequestrin (Casq) is a high capacity, low affinity Ca(2+)-binding protein, critical for Ca(2+)-buffering in cardiac and skeletal muscle sarcoplasmic reticulum. All vertebrates have multiple genes encoding for different Casq isoforms. Increasing interest has been focused on mammalian and human Casq
Kathy Ye Morgan et al.
Journal of tissue engineering and regenerative medicine, 11(2), 342-353 (2014-06-12)
Mechanical stimulation has been used extensively to improve the function of cardiac engineered tissue, as it mimics the physical environment in which the tissue is situated during normal development. However, previous mechanical stimulation has been carried out under a constant
Calsequestrins in skeletal and cardiac muscle from adult Danio rerio
Furlan S, et al.
Journal of Muscle Research and Cell Motility, 37(1-2), 27-39 (2016)
Lauren D Black et al.
Tissue engineering. Part A, 15(10), 3099-3108 (2009-04-03)
A high-potential therapy for repairing the heart post-myocardial infarction is the implantation of tissue-engineered myocardium. While several groups have developed constructs that mimic the aligned structure of the native myocardium, to date no one has investigated the particular functional benefits
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