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P60205

Sigma-Aldrich

2-Pyridinealdoxime methiodide

99%

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Synonym(s):
2-PAM, 2-PAM iodide, Pralidoxime iodide
Empirical Formula (Hill Notation):
C7H11IN2O
CAS Number:
94-63-3
Molecular Weight:
266.08
EC Number:
202-349-6
MDL number:
MFCD00011982
PubChem Substance ID:
24898726
NACRES:
NA.22

Quality Level

200

Assay

99%

form

solid

mp

220 °C (dec.) (lit.)

storage temp.

2-8°C

SMILES string

[I-].C[n+]1ccccc1\C=N\O

InChI

1S/C7H8N2O.HI/c1-9-5-3-2-4-7(9)6-8-10;/h2-6H,1H3;1H

InChI key

QNBVYCDYFJUNLO-UHFFFAOYSA-N

Gene Information

human ... ACHE(43)

Related Categories

Pictograms

Exclamation mark
GHS07

Signal Word

Warning

Hazard Statements

H302

Precautionary Statements

P264 - P270 - P301 + P312 - P501

Hazard Classifications

Acute Tox. 4 Oral

Storage Class Code

11 - Combustible Solids

WGK

WGK 3

Flash Point(F)

Not applicable

Flash Point(C)

Not applicable

Personal Protective Equipment

dust mask type N95 (US), Eyeshields, Gloves

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Lorise C Gahring et al.
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Kevin G McGarry et al.
Toxicological sciences : an official journal of the Society of Toxicology, 174(1), 124-132 (2019-12-28)
Organophosphorus (OP) compounds, which include insecticides and chemical warfare nerve agents (CWNAs) such as sarin (GB) and VX, continue to be a global threat to both civilian and military populations. It is widely accepted that cholinesterase inhibition is the primary
Qiqi Zheng et al.
Analytica chimica acta, 982, 78-83 (2017-07-25)
An electrochemical method based on non-enzymatic inhibition for the determination of organophosphorus pesticide (OPPs) using pralidoxime chloride (PAM-Cl) as a universal electrochemical probe was reported. Cyclic voltammetry was performed to characterize the redox properties of pralidoxime and OPPs. Differential pulse
Eduardo A González et al.
Archives of toxicology, 94(6), 2149-2162 (2020-04-19)
Organophosphate (OP) threat agents can trigger seizures that progress to status epilepticus, resulting in persistent neuropathology and cognitive deficits in humans and preclinical models. However, it remains unclear whether patients who do not show overt seizure behavior develop neurological consequences.
Kimberly D Spradling et al.
Journal of neuroinflammation, 8, 84-84 (2011-07-23)
Although the acute toxicity of organophosphorus nerve agents is known to result from acetylcholinesterase inhibition, the molecular mechanisms involved in the development of neuropathology following nerve agent-induced seizure are not well understood. To help determine these pathways, we previously used
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