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Sigma-Aldrich

Anti-Plasminogen/Angiostatin Antibody, clone GMA-013

clone GMA-013, Upstate®, from mouse

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eCl@ss:
32160702
NACRES:
NA.41

biological source

mouse

Quality Level

100

antibody form

purified immunoglobulin

antibody product type

primary antibodies

clone

GMA-013, monoclonal

species reactivity

human

manufacturer/tradename

Upstate®

technique(s)

ELISA: suitable
western blot: suitable

isotype

IgM

NCBI accession no.

NM_000301

UniProt accession no.

P00747

shipped in

dry ice

target post-translational modification

unmodified

Gene Information

human ... PLG(5340)

Specificity

Plasminogen (kringles 1-3)

Immunogen

Purified human plasminogen

Application

Research Category
Inflammation & Immunology
Research Sub Category
Inflammation & Autoimmune Mechanisms
This Anti-Plasminogen/Angiostatin Antibody, clone GMA-013 is validated for use in ELISA, WB for the detection of Plasminogen/Angiostatin.

Quality

routinely evaluated by immunoblot on human plasminogen

Target description

Mr 88kDa

Physical form

DEAE purified
Format: Purified

Storage and Stability

stable 2 years at -20°C from date of shipment

Legal Information

UPSTATE is a registered trademark of Merck KGaA, Darmstadt, Germany

Disclaimer

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

Storage Class Code

11 - Combustible Solids

WGK

WGK 3

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Angiostatin and anti-angiogenic therapy in human disease.
Wahl, Miriam L, et al.
Recent Progress in Hormone Research, 59, 73-104 (2004)
Christopher D Barrett et al.
The journal of trauma and acute care surgery, 83(6), 1053-1061 (2017-08-25)
A subset of trauma patients undergo fibrinolysis shutdown rather than pathologic hyperfibrinolysis, contributing to organ failure. The molecular basis for fibrinolysis shutdown in trauma is incompletely understood. Elastase released from primed/activated human neutrophils (HNE) has historically been described as fibrin(ogen)olytic.

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