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Merck
CN

36970

Bromodichloromethane

amylene stabilized, analytical standard

Synonym(s):

Dichlorobromomethane

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About This Item

Linear Formula:
BrCHCl2
CAS Number:
Molecular Weight:
163.83
EC Number:
200-856-7
UNSPSC Code:
41116107
PubChem Substance ID:
Beilstein/REAXYS Number:
1697005
MDL number:
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InChI key

FMWLUWPQPKEARP-UHFFFAOYSA-N

InChI

1S/CHBrCl2/c2-1(3)4/h1H

SMILES string

ClC(Cl)Br

grade

analytical standard

quality

amylene stabilized

shelf life

limited shelf life, expiry date on the label

technique(s)

HPLC: suitable, gas chromatography (GC): suitable

refractive index

n20/D 1.497 (lit.)

bp

87 °C (lit.)

mp

−55 °C (lit.)

density

1.98 g/mL at 25 °C (lit.)

application(s)

environmental

format

neat

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Application

Refer to the product′s Certificate of Analysis for more information on a suitable instrument technique. Contact Technical Service for further support.

signalword

Danger

Hazard Classifications

Acute Tox. 4 Oral - Carc. 2 - Eye Dam. 1 - Skin Irrit. 2 - STOT SE 3

target_organs

Respiratory system

Storage Class

10 - Combustible liquids

wgk

WGK 3

flash_point_f

Not applicable

flash_point_c

Not applicable

ppe

Eyeshields, Faceshields, Gloves, type ABEK (EN14387) respirator filter


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Richard J Summerhayes et al.
Epidemiology (Cambridge, Mass.), 23(1), 15-22 (2011-12-14)
Trihalomethanes in drinking water have been associated with higher occurrence of small-for-gestational-age (SGA) births, although results have been inconsistent. We geocoded residential address for mother of live, singleton, term births to 33 water distribution systems in a large metropolitan area
S R Bielmeier et al.
Toxicology in vitro : an international journal published in association with BIBRA, 21(5), 919-928 (2007-03-09)
Bromodichloromethane (BDCM), a drinking water disinfection by-product, causes pregnancy loss, i.e. full-litter resorption, in F344 rats when treated during the luteinizing hormone (LH)-dependent period. This effect is associated with reduced maternal serum progesterone (P) and LH levels, suggesting that BDCM
Ratanesh Kumar Seth et al.
Toxicology and applied pharmacology, 274(1), 42-54 (2013-11-12)
Environmental toxins induce a novel CYP2E1/leptin signaling axis in liver. This in turn activates a poorly characterized innate immune response that contributes to nonalcoholic steatohepatitis (NASH) progression. To identify the relevant subsets of T-lymphocytes in CYP2E1-dependent, environment-linked NASH, we utilized
Suvarthi Das et al.
American journal of physiology. Gastrointestinal and liver physiology, 305(12), G950-G963 (2013-10-26)
Recent studies indicate that metabolic oxidative stress, autophagy, and inflammation are hallmarks of nonalcoholic steatohepatitis (NASH) progression. However, the molecular mechanisms that link these important events in NASH remain unclear. In this study, we investigated the mechanistic role of purinergic
Suvarthi Das et al.
Toxicology and applied pharmacology, 269(3), 297-306 (2013-02-27)
Today's developed world faces a major public health challenge in the rise in the obese population and the increased incidence in fatty liver disease. There is a strong association among diet induced obesity, fatty liver disease and development of nonalcoholic

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