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Merck
CN

45305

Dicrotophos

PESTANAL®, analytical standard

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About This Item

Empirical Formula (Hill Notation):
C8H16NO5P
CAS Number:
141-66-2
Molecular Weight:
237.19
UNSPSC Code:
41116107
NACRES:
NA.24
PubChem Substance ID:
329756631
EC Number:
205-494-3
Beilstein/REAXYS Number:
1880084
MDL number:
MFCD00072499

Key Documents

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InChI key

VEENJGZXVHKXNB-VOTSOKGWSA-N

InChI

1S/C8H16NO5P/c1-7(6-8(10)9(2)3)14-15(11,12-4)13-5/h6H,1-5H3/b7-6+

SMILES string

COP(=O)(OC)O\C(C)=C\C(=O)N(C)C

grade

analytical standard

product line

PESTANAL®

shelf life

limited shelf life, expiry date on the label

technique(s)

HPLC: suitable, gas chromatography (GC): suitable

application(s)

agriculture
environmental

format

neat

storage temp.

2-8°C

Quality Level

100

Related Categories

Application

Refer to the product′s Certificate of Analysis for more information on a suitable instrument technique. Contact Technical Service for further support.

Other Notes

Find a digital Reference Material for this product available on our online platform ChemisTwin® for NMR. You can use this digital equivalent on ChemisTwin® for your sample identity confirmation and compound quantification (with digital external standard). An NMR spectrum of this substance can be viewed and an online comparison against your sample can be performed with a few mouseclicks. Learn more here and start your free trial.

Legal Information

PESTANAL is a registered trademark of Merck KGaA, Darmstadt, Germany

pictograms

Skull and crossbonesEnvironment
GHS06,GHS09

signalword

Danger

Hazard Classifications

Acute Tox. 1 Inhalation - Acute Tox. 2 Dermal - Acute Tox. 2 Oral - Aquatic Acute 1 - Aquatic Chronic 1

Storage Class

6.1A - Combustible, acute toxic Cat. 1 and 2 / very toxic hazardous materials

wgk

WGK 3

flash_point_f

Not applicable

flash_point_c

Not applicable

ppe

Eyeshields, Faceshields, Gloves, type ABEK (EN14387) respirator filter

Regulatory Information

剧毒化学品
危险化学品
农药列管产品
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Certificates of Analysis (COA)

Lot/Batch Number
BCCM0143
BCCH2359
BCBV7705

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Dietrich E Lorke et al.
Current pharmaceutical design, 23(23), 3432-3439 (2016-11-02)
Reversible cholinesterase inhibitors, when given prophylactically before exposure to organophosphates, are able to decrease organophosphate-induced mortality. However, the efficacy of pyridostigmine, the only pre-treatment substance approved by the US Federal Drug Administration, is unsatisfactory. In search of a better prophylactic
T Markovska et al.
The International journal of biochemistry, 25(2), 253-257 (1993-02-01)
1. Treatment of rats with carbicron induced a reduction of the phospholipids in both microsomal and plasma membranes. 2. A decrease of the structural order parameter (SDPH) and an increase of the pyrene excimer-to-monomer fluorescence ratio (IE/IM) was also observed
M Misawa et al.
Journal of toxicology and environmental health, 10(4-5), 551-563 (1982-10-01)
Teratogenic effects of two organophosphate insecticides, diazinon and dicrotophos, were investigated in regard to skeletal development, particularly of the extremities and vertebrae. Cartilage and calcified bone were examined with alcian blue and alizarin red S staining techniques, respectively, in chick
J E Snawder et al.
Journal of environmental science and health. Part. B, Pesticides, food contaminants, and agricultural wastes, 24(3), 205-218 (1989-06-01)
Four organophosphorus insecticides and the active metabolites of two phosphorothionate insecticides were tested for their toxic and teratogenic effects on embryos of Xenopus laevis. All compounds caused dose-dependent developmental defects, such as abnormal pigmentation, abnormal gut development, notochordal defects and
E Yanev et al.
Comparative biochemistry and physiology. C, Comparative pharmacology and toxicology, 103(3), 553-555 (1992-11-01)
1. Intoxication of rats with carbicron (O-([2-butenoic acid)-N,N-dimethylamide-3-yl]-O,O-dimethylphosphate) induced a reduction of the total phospholipids and phosphatidylcholine in lung alveolar surfactant. 2. The lipid transfer protein activity was inhibited due to carbicron treatment. 3. No alterations were observed in phospholipase
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