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Merck
CN

D3190

Dithiobiuret

97%, solid

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About This Item

CAS Number:
NACRES:
NA.77
PubChem Substance ID:
UNSPSC Code:
12352200
EC Number:
208-784-8
MDL number:
Assay:
97%
Form:
solid
Quality level:
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InChI key

JIRRNZWTWJGJCT-UHFFFAOYSA-N

InChI

1S/C2H5N3S2/c3-1(6)5-2(4)7/h(H5,3,4,5,6,7)

SMILES string

NC(=S)NC(N)=S

assay

97%

form

solid

Quality Level

General description

Paralytic agent.

pictograms

Skull and crossbones

signalword

Danger

Hazard Classifications

Acute Tox. 1 Inhalation - Acute Tox. 2 Dermal - Acute Tox. 2 Oral

Storage Class

6.1A - Combustible acute toxic Cat. 1 and 2 / very toxic hazardous materials

wgk

WGK 3

flash_point_f

Not applicable

flash_point_c

Not applicable

ppe

Eyeshields, Faceshields, Gloves, type P3 (EN 143) respirator cartridges

Regulatory Information

新产品
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Z Sahenk
Acta neuropathologica, 81(2), 141-147 (1990-01-01)
It has been well established that 2,4-dithiobiuret (DTB) intoxication in rats produces a rapidly progressive hindlimb paralysis within days. The cause of this has, until recently, been explained on the basis of a physiological abnormality that involves a prejunctional impairment
K M Crofton et al.
Fundamental and applied toxicology : official journal of the Society of Toxicology, 16(3), 469-481 (1991-04-01)
2,4-Dithiobiuret (DTB) exposure causes a delayed onset muscle weakness in rats that has been attributed to depressed neuromuscular transmission. The present study compares the effects of DTB on both sensory and motor function in rats. Adult male Long-Evans hooded rats
Dithiobiuret-induced muscle weakness in rats: evidence for a prejunctional effect.
W D Atchison et al.
Neurotoxicology, 3(1), 44-54 (1982-07-01)
K D Williams et al.
Toxicology and applied pharmacology, 91(2), 212-221 (1987-11-01)
To evaluate the hypothesis that depressed neuromuscular transmission causes dithiobiuret (DTB)-induced muscle weakness in rats, the temporal development of impaired treadmill performance and deficits in the nerve-elicited muscle contractions were compared during daily treatment with the toxicant (DTB, 1 mg/kg/day
W D Atchison et al.
Experimental neurology, 85(1), 63-68 (1984-07-01)
Urinary excretion of porphyrin precursors delta-aminolevulinic acid (ALA) and porphobilinogen (PBG) and total porphyrins was measured during intoxication of rats with 2,4-dithiobiuret (DTB), a chemical which produces delayed-onset neuromuscular weakness, in an attempt to ascertain whether or not DTB poisoning

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