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EHU007271

MISSION® esiRNA

targeting human MTCH2

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About This Item

NACRES:
NA.51
UNSPSC Code:
41105324

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Product Name

MISSION® esiRNA, targeting human MTCH2

description

Powered by Eupheria Biotech

product line

MISSION®

form

lyophilized powder

esiRNA cDNA target sequence

CACATTGCCAGTATCGATGGGAGGCGCGGGTTGTTCACAGGCTTAACTCCAAGACTGTGTTCGGGAGTCCTTGGAACTGTGGTCCATGGTAAAGTTTTACAGCATTACCAGGAGAGTGACAAGGGTGAGGAGTTAGGACCTGGAAATGTACAGAAAGAAGTCTCATCTTCCTTTGACCACGTTATCAAGGAGACAACTCGAGAGATGATCGCTCGTTCTGCTGCTACCCTCATCACACATCCCTTCCATGTGATCACTCTGAGATCTATGGTACAGTTCATTGGCAGAGAATCCAAGTACTGTGGACTTTGTGATTCCATAATAACCATCTATCGGGAAGAGGGCATTCTAGGATTTTTCGCGGGTCTTGTTCCTCGCCTTCTAGGTGACATCCTTTCTTTGTGGCTGTGTAACTCACTGGCCTACCTCGTCAAT

Ensembl | human accession no.

ENSG00000109919

NCBI accession no.

NM_014342

shipped in

ambient

storage temp.

−20°C

Quality Level

100

Gene Information

human ... MTCH2(23788), MTCH2(23788)

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General description

MISSION® esiRNA are endoribonuclease prepared siRNA. They are a heterogeneous mixture of siRNA that all target the same mRNA sequence. These multiple silencing triggers lead to highly-specific and effective gene silencing.

For additional details as well as to view all available esiRNA options, please visit SigmaAldrich.com/esiRNA.

Legal Information

MISSION is a registered trademark of Merck KGaA, Darmstadt, Germany

Storage Class

10 - Combustible liquids

flash_point_f

Not applicable

flash_point_c

Not applicable

Regulatory Information

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Dilshad H Khan et al.
Blood, 136(1), 81-92 (2020-04-17)
Through a clustered regularly insterspaced short palindromic repeats (CRISPR) screen to identify mitochondrial genes necessary for the growth of acute myeloid leukemia (AML) cells, we identified the mitochondrial outer membrane protein mitochondrial carrier homolog 2 (MTCH2). In AML, knockdown of
Qiuyun Yuan et al.
Molecular medicine (Cambridge, Mass.), 27(1), 7-7 (2021-01-30)
Malignant glioma exerts a metabolic shift from oxidative phosphorylation (OXPHOs) to aerobic glycolysis, with suppressed mitochondrial functions. This phenomenon offers a proliferation advantage to tumor cells and decrease mitochondria-dependent cell death. However, the underlying mechanism for mitochondrial dysfunction in glioma

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MISSION esiRNA

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