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M1555

MAHMA NONOate

Name: MAHMA NONOate
Brand: SIGMA

Synonym(s):

6-(2-Hydroxy-1-methyl-2-nitrosohydrazino)-N-methyl-1-hexanamine, NOC-9

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About This Item

Empirical Formula (Hill Notation):

C₈H₂₀N₄O₂

CAS Number:

146724-86-9

Molecular Weight:

204.27

NACRES:

NA.25

PubChem Substance ID:

24896668

UNSPSC Code:

12352106

MDL number:

MFCD01310509

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Properties

Quality Level

200

shipped in

dry ice

storage temp.

−70°C

SMILES string

[H][N+]([H])(C)CCCCCCN(C)N([O-])N=O

InChI

1S/C8H19N4O2/c1-9-7-5-3-4-6-8-11(2)12(14)10-13/h9H,3-8H2,1-2H3/q-1/p+1

InChI key

WASRCWNCCFPNEJ-UHFFFAOYSA-O

Description

Application

NO donor

Biochem/physiol Actions

NO release: t_1/2 = 3 min (phosphate buffer at 22° C)

Storage Class

11 - Combustible Solids

wgk

WGK 3

flash_point_f

Not applicable

flash_point_c

Not applicable

ppe

Eyeshields, Gloves, type N95 (US)

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Articles

Sensor Dyes

Nitric oxide (NO) as a signal transporter in neurons, endothelial cells and in the immune system.

感应染料

一氧化氮 (NO) 是神经元、内皮细胞和免疫系统中的信号转运分子。

Exposure of Stored Packed Erythrocytes to Nitric Oxide Prevents Transfusion-associated Pulmonary Hypertension.

Stefan Muenster et al.

Anesthesiology, 125(5), 952-963 (2016-10-19)

Transfusion of packed erythrocytes stored for a long duration is associated with increased pulmonary arterial pressure and vascular resistance. Prolonged storage decreases erythrocyte deformability, and older erythrocytes are rapidly removed from the circulation after transfusion. The authors studied whether treating

Nitric oxide inhibition of tobacco catalase and ascorbate peroxidase.

D Clark et al.

Molecular plant-microbe interactions : MPMI, 13(12), 1380-1384 (2000-12-06)

We used a variety of nitric oxide (NO) donors to demonstrate that NO inhibits the activities of tobacco catalase and ascorbate peroxidase (APX). This inhibition appears to be reversible because removal of the NO donor led to a significant recovery

Prevention of nitric oxide-induced neuronal injury through the modulation of independent pathways of programmed cell death.

S H Lin et al.

Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism, 20(9), 1380-1391 (2000-09-20)

Neuronal injury may be dependent upon the generation of the free radical nitric oxide (NO) and the subsequent induction of programmed cell death (PCD). Although the nature of this injury may be both preventable and reversible, the underlying mechanisms that

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Global Trade Item Number

SKU	GTIN
M1555-50MG	04061834040903