RAB0012
Phospho-Akt (pSer473) / pan-Akt ELISA Kit
for detection of human, mouse or rat phospho-akt (pSer473) and pan-akt in cell and tissue lysates
Synonym(s):
Akt ELISA Kit
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About This Item
UNSPSC Code:
41116158
NACRES:
NA.32
species reactivity
human, mouse, rat
packaging
kit of 96 wells (12 strips x 8 wells)
technique(s)
ELISA: suitable
capture ELISA: suitable
input
sample type: human cell lysate
sample type tissue lysate
detection method
colorimetric
shipped in
wet ice
storage temp.
−20°C
Gene Information
human ... AKT1(207)
General description
The Phospho-Akt (pSer473) ELISA (Enzyme-Linked Immunosorbent Assay) kit is a very rapid, convenient and sensitive assay kit that can monitor the activation or function of important biological pathways in cell lysates.
Immunogen
Akt (pSer473) synthetic phosphopeptide, Recombinant Human EGFR
Application
For research use only. Not for use in diagnostic procedures.
Please refer to the attached General ELISA KIT Procedure (sandwich, competitive & Indirect ELISA)
Please refer to the attached General ELISA KIT Procedure (sandwich, competitive & Indirect ELISA)
Kit Components Also Available Separately
Product No.
Description
SDS
- RABTMB3ELISA Colorimetric TMB Reagent (HRP Substrate, Item H)
Signal Word
Warning
Hazard Statements
Precautionary Statements
Hazard Classifications
Met. Corr. 1
Storage Class Code
8B - Non-combustible corrosive hazardous materials
WGK
WGK 3
Flash Point(F)
Not applicable
Flash Point(C)
Not applicable
Regulatory Information
低风险生物材料
常规特殊物品
易制毒化学品（3类）
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Fatima Maqoud et al.
International journal of molecular sciences, 19(8) (2018-08-22)
The effects of Ca2+-activated K⁺ (BK) channel modulation by Paxilline (PAX) (10-7⁻10-4 M), Iberiotoxin (IbTX) (0.1⁻1 × 10-6 M) and Resveratrol (RESV) (1⁻2 × 10-4 M) on cell cycle and proliferation, AKT1pSer473 phosphorylation, cell diameter, and BK currents were investigated
Molly Stanley et al.
The Journal of neuroscience : the official journal of the Society for Neuroscience, 36(46), 11704-11715 (2016-11-18)
Hyperinsulinemia is a risk factor for late-onset Alzheimer's disease (AD). In vitro experiments describe potential connections between insulin, insulin signaling, and amyloid-β (Aβ), but in vivo experiments are needed to validate these relationships under physiological conditions. First, we performed hyperinsulinemic-euglycemic
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