biological source
rabbit
Quality Level
conjugate
unconjugated
antibody form
affinity isolated antibody
antibody product type
primary antibodies
clone
polyclonal
form
buffered aqueous solution
mol wt
antigen 23 kDa
species reactivity
mouse, human, rat
technique(s)
western blot: suitable
UniProt accession no.
shipped in
dry ice
storage temp.
−20°C
target post-translational modification
unmodified
Gene Information
human ... AANAT(15)
mouse ... Aanat(11298)
rat ... Aanat(25120)
Immunogen
synthetic phosphopeptide corresponding to amino-terminal amino acid residues 24-33 of mouse arylalkylamine N-acetyltransferase conjugated to KLH.
Physical form
Solution in phosphate buffered saline, containing 1 mg/ml bovine serum albumin and 0.05% sodium azide
Preparation Note
The non-phospho-specific antibody was isolated on non-phospho-peptide-coupled resin.
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Storage Class Code
10 - Combustible liquids
WGK
WGK 3
Flash Point(F)
Not applicable
Flash Point(C)
Not applicable
Regulatory Information
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Marco A Pires-Lapa et al.
International journal of molecular sciences, 19(8) (2018-07-28)
Melatonin (5-methoxy-N-acetylserotonin), the pineal hormone, is also synthesized by immune-competent cells. The pineal hormone signals darkness, while melatonin synthesized on demand by activated macrophages at any hour of the day acts locally, favoring regulatory/tolerant phenotypes. Activation of β-adrenoceptors in pinealocytes
Marlina O Córdoba-Moreno et al.
Scientific reports, 10(1), 4799-4799 (2020-03-18)
Daily oscillation of the immune system follows the central biological clock outputs control such as melatonin produced by the pineal gland. Despite the literature showing that melatonin is also synthesized by macrophages and T lymphocytes, no information is available regarding
Claudia Emanuele Carvalho-Sousa et al.
Journal of pineal research, 68(3), e12636-e12636 (2020-02-12)
Environmental pollution in the form of particulate matter <2.5 μm (PM2.5 ) is a major risk factor for diseases such as lung cancer, chronic respiratory infections, and major cardiovascular diseases. Our goal was to show that PM2.5 eliciting a proinflammatory response
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