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Merck
CN

SAB1305552

Anti-MAP1LC3A antibody

mouse monoclonal, 166AT1234

Synonym(s):

Autophagy-related protein LC3 A, Autophagy-related ubiquitin-like modifier LC3 A, MAP1 light chain 3-like protein 1, MAP1LC3A, Microtubule-associated proteins 1A/1B light chain 3A

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About This Item

UNSPSC Code:
12352203
NACRES:
NA.41
Clone:
166AT1234, monoclonal
Species reactivity:
rat, mouse, human
Application:
immunofluorescence
immunohistochemistry
western blot
Technique(s):
immunofluorescence: 1:25
immunohistochemistry: 1:50-1:100
western blot: 1:1000
Citations:
7
Uniprot accession no.:
Q9H492

Key Documents

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Product Name

MONOCLONAL ANTI-LC3 (APG8) antibody produced in mouse, clone 166AT1234, IgG fraction of antiserum, buffered aqueous solution

Quality Level

100

biological source

mouse

antibody form

IgG fraction of antiserum

antibody product type

primary antibodies

clone

166AT1234, monoclonal

form

buffered aqueous solution

mol wt

14272 Da

species reactivity

rat, mouse, human

technique(s)

immunofluorescence: 1:25
immunohistochemistry: 1:50-1:100
western blot: 1:1000

isotype

IgG1κ

NCBI accession no.

NP_115903.1

UniProt accession no.

Q9H492

shipped in

wet ice

storage temp.

−20°C

target post-translational modification

unmodified

Gene Information

human ... MAP1LC3A(84557)

Disclaimer

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

Physical form

Supplied in PBS with 0.09% (W/V) sodium azide

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Storage Class

10 - Combustible liquids

flash_point_f

Not applicable

flash_point_c

Not applicable

Regulatory Information

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Certificates of Analysis (COA)

Lot/Batch Number
SG190329AA
SD100625
SD110414A
SDG090127
SG100621AB

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Matteo Bordoni et al.
Cells, 11(8) (2022-04-24)
Mitochondria alterations are present in tissues derived from patients and animal models, but no data are available for peripheral blood mononuclear cells (PBMCs) of ALS patients. This work aims to investigate mitophagy in PBMCs of sporadic (sALS) patients and how
Hong Ding et al.
Journal of asthma and allergy, 17, 717-731 (2024-08-06)
Accumulating evidence indicates that oxidative stress and inflammation are the pathological basis of allergic diseases. Inhibition of NOD-like receptor family pyrin domain-containing 3 (NLRP3) inflammasome could ameliorate allergic rhinitis (AR). Here, we explored the effects and mechanisms that underlie NLRP3
Xiaoming Shu et al.
Molecular medicine reports, 16(2), 1180-1188 (2017-06-07)
Peripheral blood T lymphocytopenia has previously been identified in polymyositis/dermatomyositis (PM/DM) patients. Therefore, the present study aimed to examine the potential role of autophagy in peripheral blood T cell survival in PM/DM patients. Transmission electron microscopy was used to detect
Maomao Sun et al.
Frontiers in immunology, 12, 685523-685523 (2021-08-03)
Recent studies have shown that autophagy upregulation can attenuate sepsis-induced acute kidney injury (SAKI). The tumor suppressor p53 has emerged as an autophagy regulator in various forms of acute kidney injury (AKI). Our previous studies showed that p53 acetylation exacerbated
Rongrong Hua et al.
Journal of cellular biochemistry, 120(9), 15915-15923 (2019-05-14)
The sequential reactivation of mechanistic target of rapamycin (mTOR) inhibited autophagic flux in neurons exposed to oxygen-glucose deprivation/reperfusion (OGD/R), which was characterized by reduction of autophagosome formation and restriction of autolysosome degradation. However, its detailed molecular mechanism was still unknown.
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