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SAB4501304

Sigma-Aldrich

Anti-NMDAR2A antibody produced in rabbit

affinity isolated antibody

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Synonym(s):
GRIN2A, Glutamate [NMDA] receptor subunit ε 1 precursor, N-methyl D-aspartate receptor subtype 2A, N-methyl-D-aspartate receptor subtype 2A, NMDAR2AGlutamate [NMDA] receptor subunit ε 1
NACRES:
NA.41

biological source

rabbit

Quality Level

100

conjugate

unconjugated

antibody form

affinity isolated antibody

antibody product type

primary antibodies

clone

polyclonal

form

buffered aqueous solution

mol wt

antigen 165 kDa

species reactivity

rat, mouse, human

concentration

~1 mg/mL

technique(s)

ELISA: 1:1000
immunofluorescence: 1:100-1:500
immunohistochemistry: 1:50-1:100

NCBI accession no.

Q12879

UniProt accession no.

Q12879

shipped in

wet ice

storage temp.

−20°C

target post-translational modification

unmodified

Gene Information

human ... GRIN2A(2903)

General description

Anti-NMDAR2A Antibody detects endogenous levels of total NMDAR2A protein.

Immunogen

The antiserum was produced against synthesized peptide derived from human NMDAR2A/B.

Immunogen Range: 1216-1265

Features and Benefits

Evaluate our antibodies with complete peace of mind. If the antibody does not perform in your application, we will issue a full credit or replacement antibody. Learn more.

Physical form

Rabbit IgG in phosphate buffered saline (without Mg2+ and Ca2+), pH 7.4, 150mM NaCl, 0.02% sodium azide and 50% glycerol.

Disclaimer

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

Storage Class Code

10 - Combustible liquids

WGK

nwg

Flash Point(F)

Not applicable

Flash Point(C)

Not applicable

Regulatory Information

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Wei-Yi Gong et al.
Frontiers in aging neuroscience, 9, 123-123 (2017-05-30)
Many factors impact cognitive impairment; however, the effects of chronic pain and the mechanisms underlying these effects on cognitive impairment are currently unknown. Here we tested the hypothesis that chronic pain accelerates the transition from normal cognition to mild cognitive
Przemysław Duda et al.
Aging, 10(7), 1682-1697 (2018-07-22)
Aging is believed to be the result of alterations of protein expression and accumulation of changes in biomolecules. Although there are numerous reports demonstrating changes in protein expression in brain during aging, only few of them describe global changes at

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