316970
氢氧化铈(IV)
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关于此项目
线性分子式:
Ce(OH)4
化学文摘社编号:
分子量:
208.15
NACRES:
NA.23
PubChem Substance ID:
UNSPSC Code:
12352302
EC Number:
234-599-7
MDL number:
Form:
chunks
form
chunks
InChI key
WTVAYLQYAWAHAX-UHFFFAOYSA-J
InChI
1S/Ce.4H2O/h;4*1H2/q+4;;;;/p-4
SMILES string
O[Ce](O)(O)O
reaction suitability
core: cerium
reagent type: catalyst
concentration
≥47.1% Ce (after ignition to oxide, gravimetric)
Quality Level
存储类别
11 - Combustible Solids
wgk
WGK 3
flash_point_f
Not applicable
flash_point_c
Not applicable
ppe
dust mask type N95 (US), Eyeshields, Gloves
Mo Dan et al.
Nanomedicine (London, England), 7(1), 95-110 (2011-12-24)
Characterize different sized ceria-engineered nanomaterial (ENM) distribution in, and clearance from, blood (compared to the cerium ion) following intravenous infusion. Cerium (Ce) was quantified in whole blood, serum and clot (the formed elements) up to 720 h. Traditional pharmacokinetic modeling
Development of nonaluminum phosphate binder hydrous cerium oxide (Ce alpha (OH)4-2 alpha . nH2O).
S Nakagawa et al.
Transactions - American Society for Artificial Internal Organs, 31, 155-159 (1985-01-01)
Karen E Keith et al.
Microbiology (Reading, England), 155(Pt 4), 1004-1015 (2009-04-01)
Burkholderia cenocepacia causes chronic lung infections in patients suffering from cystic fibrosis and chronic granulomatous disease. We have previously shown that B. cenocepacia survives intracellularly in macrophages within a membrane vacuole (BcCV) that delays acidification. Here, we report that after
W M Frederiks et al.
Free radical biology & medicine, 22(1-2), 241-248 (1997-01-01)
Superoxide anion radicals have been implicated in a variety of pathological processes. Under physiological conditions, superoxide dismutase (SOD) is effectively able to disproportionate superoxide anions into hydrogen peroxide and dioxygen. Until now, no techniques have been available to localize SOD
J Slezak et al.
The American journal of pathology, 147(3), 772-781 (1995-09-01)
Active oxygen species including hydrogen peroxide (H2O2) play a major role in ischemia-reperfusion injury. In the present study, changes in myocardial H2O2 content as well as its subcellular distribution were examined in rat hearts subjected to ischemia-reperfusion. Isolated perfused rat
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