609358
氰酸钾-15N
≥98 atom % 15N, ≥95% (CP)
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关于此项目
线性分子式:
KOC15N
化学文摘社编号:
分子量:
82.11
UNSPSC Code:
12352118
PubChem Substance ID:
MDL number:
Isotopic purity:
≥98 atom % 15N
Assay:
≥95% (CP)
Mass shift:
M+1
Form:
solid
InChI
1S/CHNO.K/c2-1-3;/h3H;/q;+1/p-1/i2+1;
SMILES string
[K+].[O-]C#[15N]
InChI key
GKKCIDNWFBPDBW-CGOMOMTCSA-M
isotopic purity
≥98 atom % 15N
assay
≥95% (CP)
form
solid
mass shift
M+1
Packaging
This product may be available from bulk stock and can be packaged on demand. For information on pricing, availability and packaging, please contact Stable Isotopes Customer Service.
Grégoire Danger et al.
Amino acids, 42(6), 2331-2341 (2011-07-20)
The reaction of cyanate with C-terminal carboxyl groups of peptides in aqueous solution was considered as a potential pathway for the abiotic formation of peptide bonds under the condition of the primitive Earth. The catalytic effect of dicarboxylic acids on
Jirí Verner et al.
Molecules (Basel, Switzerland), 11(1), 34-42 (2007-10-27)
The reactivity of alicyclic ketazines in criss-cross cycloadditions was investigated. They react with potassium cyanate and ammonium thiocyanate in the presence of acetic acid to form spirocyclic perhydro[1,2,4]triazolo[1,2-alpha][1,2,4]triazole-1,5-diones and perhydro[1,2,4]triazolo[1,2-alpha][1,2,4]triazole-1,5-dithiones, respectively, in relatively high yields.
George S Espie et al.
Journal of bacteriology, 189(3), 1013-1024 (2006-11-24)
The cyanobacteria Synechococcus elongatus strain PCC7942 and Synechococcus sp. strain UTEX625 decomposed exogenously supplied cyanate (NCO-) to CO2 and NH3 through the action of a cytosolic cyanase which required HCO3- as a second substrate. The ability to metabolize NCO- relied
J Zambonin et al.
Journal of neuroscience methods, 192(1), 115-120 (2010-07-28)
Mitochondrial defects have been implicated in the degeneration of axons in a number of CNS disorders, including multiple sclerosis. Uniquely, mitochondria harbor the only non-nuclear DNA (mitochondrial DNA or mtDNA), which encodes functionally important subunits of the respiratory chain. The
R M Satpute et al.
Neurotoxicology, 29(1), 170-178 (2007-12-15)
Cyanide is a rapidly acting neurotoxin that inhibits cellular respiration and energy metabolism leading to histotoxic hypoxia. This results in the dissipation of mitochondrial membrane potential (MMP) accompanied by decreased cellular ATP content which in turn is responsible for increased
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