609358
氰酸钾-15N
≥98 atom % 15N, ≥95% (CP)
同位素纯度
≥98 atom % 15N
方案
≥95% (CP)
表单
solid
质量偏移
M+1
SMILES字符串
[K+].[O-]C#[15N]
InChI
1S/CHNO.K/c2-1-3;/h3H;/q;+1/p-1/i2+1;
InChI key
GKKCIDNWFBPDBW-CGOMOMTCSA-M
包装
This product may be available from bulk stock and can be packaged on demand. For information on pricing, availability and packaging, please contact Stable Isotopes Customer Service.
R M Satpute et al.
Neurotoxicology, 29(1), 170-178 (2007-12-15)
Cyanide is a rapidly acting neurotoxin that inhibits cellular respiration and energy metabolism leading to histotoxic hypoxia. This results in the dissipation of mitochondrial membrane potential (MMP) accompanied by decreased cellular ATP content which in turn is responsible for increased
George S Espie et al.
Journal of bacteriology, 189(3), 1013-1024 (2006-11-24)
The cyanobacteria Synechococcus elongatus strain PCC7942 and Synechococcus sp. strain UTEX625 decomposed exogenously supplied cyanate (NCO-) to CO2 and NH3 through the action of a cytosolic cyanase which required HCO3- as a second substrate. The ability to metabolize NCO- relied
Jirí Verner et al.
Molecules (Basel, Switzerland), 11(1), 34-42 (2007-10-27)
The reactivity of alicyclic ketazines in criss-cross cycloadditions was investigated. They react with potassium cyanate and ammonium thiocyanate in the presence of acetic acid to form spirocyclic perhydro[1,2,4]triazolo[1,2-alpha][1,2,4]triazole-1,5-diones and perhydro[1,2,4]triazolo[1,2-alpha][1,2,4]triazole-1,5-dithiones, respectively, in relatively high yields.
J Zambonin et al.
Journal of neuroscience methods, 192(1), 115-120 (2010-07-28)
Mitochondrial defects have been implicated in the degeneration of axons in a number of CNS disorders, including multiple sclerosis. Uniquely, mitochondria harbor the only non-nuclear DNA (mitochondrial DNA or mtDNA), which encodes functionally important subunits of the respiratory chain. The
Sarah C Schock et al.
Brain research, 1168, 129-138 (2007-08-21)
Cortical Spreading Depression (CSD) is a well-studied model of preconditioning that provides a high degree of tolerance to a subsequent ischemic event in the brain. The present study was undertaken in order to determine whether the release of ATP during
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