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Sigma-Aldrich

3,4-二甲氧基噻吩

97%

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About This Item

经验公式（希尔记法）:
C6H8O2S
CAS Number:
51792-34-8
分子量:
144.19
MDL编号:
MFCD01096546
UNSPSC代码:
12352103
PubChem化学物质编号:
24884971
NACRES:
NA.23
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质量水平

100

方案

97%

折射率

n20/D 1.5409

沸点

100-102 °C/10-11 mmHg

密度

1.209 g/mL at 25 °C

储存温度

−20°C

SMILES字符串

COc1cscc1OC

InChI

1S/C6H8O2S/c1-7-5-3-9-4-6(5)8-2/h3-4H,1-2H3

InChI key

ZUDCKLVMBAXBIF-UHFFFAOYSA-N

相关类别

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Interferon αA from mouse

Interferon β from rat ≥95%, recombinant, expressed in CHO cells, lyophilized powder, suitable for cell culture

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Interferon β from rat

Interferon α from rat ≥95%, recombinant, expressed in bacteria, lyophilized powder, suitable for cell culture

I8657

Interferon α from rat

technique(s)

cell culture | mammalian: suitable

technique(s)

cell culture | mammalian: suitable

technique(s)

cell culture | mammalian: suitable

technique(s)

cell culture | mammalian: suitable

recombinant

expressed in E. coli

recombinant

expressed in E. coli

recombinant

expressed in CHO cells

recombinant

expressed in bacteria

assay

≥95%

assay

≥95%

assay

≥95%

assay

≥95%

biological source

mouse

biological source

mouse

biological source

rat

biological source

rat

Quality Level

200

Quality Level

200

Quality Level

200

Quality Level

200

form

buffered aqueous solution

form

buffered aqueous solution

form

lyophilized powder

form

lyophilized powder

一般描述

3,4-二甲氧基噻吩（DMOT）是一种单体和前体，其可以通过2,3-二甲氧基-1,3-丁二烯与二氯化硫在己烷介质中的闭环反应合成。它是一种寡聚噻吩，主要用于开发有机电子应用中的电活性材料。[1][2]

应用

DMOT可进行酯交换反应形成3,4-乙撑二氧噻吩（EDOT）。它可进一步聚合生成PEDOT，其可用作π共轭体系中的导电聚合物。[3][4]它可以聚合形成聚（二甲氧基噻吩），其有望用于电化学掺杂以制造储能装置。[5]
合成N2S2-N4卟啉二分体的构件，用于研究光致能量传递。[6]

象形图

Exclamation mark
GHS07

警示用语：

Warning

危险声明

H302

危险分类

Acute Tox. 4 Oral

储存分类代码

10 - Combustible liquids

WGK

WGK 3

闪点(°F)

224.1 °F - closed cup

闪点(°C)

106.7 °C - closed cup

个人防护装备

Eyeshields, Faceshields, Gloves, type ABEK (EN14387) respirator filter

法规信息

常规特殊物品
含少量动物源组分生物产品
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    分析证书(COA)

    Lot/Batch Number
    MKCV3350
    MKCL2817
    MKCK7163
    MKCC5781
    MKBX3701V

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    Vasco Marcato et al.
    Molecular and cellular biology, 36(1), 13-29 (2015-10-16)
    Rapid upregulation of interferon beta (IFN-β) expression following virus infection is essential to set up an efficient innate antiviral response. Biological roles related to the antiviral and immune response have also been associated with the constitutive production of IFN-β in
    Patrick Ejlerskov et al.
    Cell, 163(2), 324-339 (2015-10-10)
    Neurodegenerative diseases have been linked to inflammation, but whether altered immunomodulation plays a causative role in neurodegeneration is not clear. We show that lack of cytokine interferon-β (IFN-β) signaling causes spontaneous neurodegeneration in the absence of neurodegenerative disease-causing mutant proteins.
    Judith van Holten et al.
    Arthritis research & therapy, 6(3), R239-R249 (2004-05-15)
    We investigated the therapeutic potential and mechanism of action of IFN-beta protein for the treatment of rheumatoid arthritis (RA). Collagen-induced arthritis was induced in DBA/1 mice. At the first clinical sign of disease, mice were given daily injections of recombinant
    Leesa M Pennell et al.
    Immunology, 152(3), 439-450 (2017-06-25)
    CD11c+ dendritic cells (DCs) exert a critical role as antigen-presenting cells in regulating pathogenic T cells in multiple sclerosis (MS). To determine whether the therapeutic benefit of interferon-β (IFN-β) treatment for MS is in part influenced by IFN regulation of
    ENU-induced mutation in USP18 causes hyperactivation of IFN-alphabeta signalling and suppresses STAT4-induced IFN-gamma production resulting in increased susceptibility to Salmonella Typhimurium
    Richer E, et al.
    Journal of immunology (Baltimore, Md. : 1950), 185(6), 3593-3593 (2010)
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