产品名称
BRAND® 96 孔微孔板，U 形底, round bottom, non-sterile
material
polypropylene
round bottom
sterility
non-sterile
packaging
pack of 100 ea
manufacturer/tradename
BRAND 701330
size
96 well
well maximum volume
300 μm
suitability
suitable for (bacteriological assays)
binding type
non-treated surface
Features and Benefits
- BRAND® 96 孔微孔板，U 形底是一种深孔板，非常适合凝集和其他需要搅拌和洗涤样品的测定。
- 具有圆形井底，没有边缘。
- 孔口周围的凸起环可防止可能的交叉污染。
- 与一系列微孔板离心机兼容。
- 可使用合适的密封垫或自粘密封膜牢固密封。
- 用于准确识别的字母数字代码。
- 由高透明度的医用级聚丙烯（PP） 制成，有助于提高样品的可见性。
- 耐大多数溶剂，包括 DMSO、苯酚和氯仿。
General description
PP。最大上样体积300 µl。基本适配所有主要的微孔板离心机。每个孔口周围的凸起环可最大限度减少可能的交叉污染。孔板可用热封膜密封，如耐DMSO密封膜（横切），带字母数字编码。
Legal Information
BRAND is a registered trademark of BRAND GMBH + CO KG
Stephen D Carter et al.
Science advances, 6(14), eaay9572-eaay9572 (2020-04-10)
The endoplasmic reticulum (ER) is a highly dynamic network of membranes. Here, we combine live-cell microscopy with in situ cryo-electron tomography to directly visualize ER dynamics in several secretory cell types including pancreatic β-cells and neurons under near-native conditions. Using
Gefei Chen et al.
Communications biology, 3(1), 32-32 (2020-01-22)
Molecular chaperones play important roles in preventing protein misfolding and its potentially harmful consequences. Deterioration of molecular chaperone systems upon ageing are thought to underlie age-related neurodegenerative diseases, and augmenting their activities could have therapeutic potential. The dementia relevant domain
SeongJun Han et al.
Oncoimmunology, 9(1), 1737368-1737368 (2020-04-22)
Regulatory T cells are integral to the regulation of autoimmune and anti-tumor immune responses. However, several studies have suggested that changes in T cell signaling networks can result in T cells that are resistant to the suppressive effects of regulatory
D Tews et al.
Redox biology, 26, 101286-101286 (2019-08-06)
Brown adipose tissue (BAT) has been considered beneficial for metabolic health by participating in the regulation of glucose homoeostasis. The browning factors that improve glucose uptake beyond normal levels are still unknown but glucose uptake is not affected in UCP1
Rosalie Heilig et al.
Life science alliance, 3(6) (2020-04-30)
Caspase-1 drives a lytic inflammatory cell death named pyroptosis by cleaving the pore-forming cell death executor gasdermin-D (GSDMD). Gsdmd deficiency, however, only delays cell lysis, indicating that caspase-1 controls alternative cell death pathways. Here, we show that in the absence
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