L2000
钙 L -乳酸 水合物
~90%
别名:
(S)-(+)-2-羟基丙酸 半钙盐, L-乳酸 半钙盐, 肌乳酸 半钙盐
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关于此项目
线性分子式:
[CH3CH(OH)COO]2Ca · xH2O
化学文摘社编号:
分子量:
218.22 (anhydrous basis)
UNSPSC Code:
12352207
NACRES:
NA.22
PubChem Substance ID:
EC Number:
248-953-3
Beilstein/REAXYS Number:
4923285
MDL number:
产品名称
钙 L -乳酸 水合物, ~90%
InChI key
MDAVASCOAJMZHZ-NVKWYWNSSA-L
InChI
1S/2C3H6O3.Ca.H2O/c2*1-2(4)3(5)6;;/h2*2,4H,1H3,(H,5,6);;1H2/q;;+2;/p-2/t2*2-;;/m00../s1
SMILES string
O.C[C@H](O)C(=O)O[Ca]OC(=O)[C@H](C)O
assay
~90%
storage temp.
2-8°C
Quality Level
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Application
Calcium L-lactate hydrate has been used as a sole carbon and energy source, to study the sulfate reduction ability of Desulfovibrio desulfuricans under different conditions.
存储类别
11 - Combustible Solids
wgk
WGK 1
flash_point_f
Not applicable
flash_point_c
Not applicable
ppe
Eyeshields, Gloves, type N95 (US)
Marlies P Noz et al.
Stroke, 49(12), 2910-2917 (2018-12-21)
Background and Purpose- Cerebral small vessel disease (cSVD) is the major vascular cause of cognitive decline and dementia. The pathogenesis of cSVD remains largely unknown, although several studies suggest a role for systemic inflammation. In certain pathophysiological situations, monocytes can
Effects of temperature and phosphorous concentration on microbial sulfate reduction by Desulfovibrio desulfuricans.
Okabe S and Characklis WG.
Biotechnology and Bioengineering, 39(10), 1031-1042 (1992)
Factors affecting microbial sulfate reduction by Desulfovibrio desulfuricans in continuous culture: limiting nutrients and sulfide concentration.
Okabe S, et al.
Biotechnology and Bioengineering, 40(6), 725-734 (1992)
Gaël Auray et al.
Frontiers in immunology, 11, 1429-1429 (2020-08-01)
The present study investigated the transcriptomic response of porcine dendritic cells (DC) to innate stimulation in vitro and in vivo. The aim was to identify DC subset-specialization, suitable Toll-like receptor (TLR) ligands targeting plasmacytoid DC (pDC), and the DC activation
Jorge Domínguez-Andrés et al.
Cell metabolism, 29(1), 211-220 (2018-10-09)
Sepsis involves simultaneous hyperactivation of the immune system and immune paralysis, leading to both organ dysfunction and increased susceptibility to secondary infections. Acute activation of myeloid cells induced itaconate synthesis, which subsequently mediated innate immune tolerance in human monocytes. In contrast, induction
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