T82805
三苯乙烯
99%
别名:
1,1′,1′′-(1-乙烯基-2-亚基)三[苯］2,6-二氯苯酚, 1,1,2-三苯基乙烯, 1,2-二苯基乙烯基苯, 乙烯-1,1,2-三基三苯, 二苯基二联苯甲烷
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关于此项目
线性分子式:
C6H5CH=C(C6H5)2
化学文摘社编号:
分子量:
256.34
UNSPSC Code:
12352100
NACRES:
NA.22
PubChem Substance ID:
EC Number:
200-395-1
Beilstein/REAXYS Number:
1867462
MDL number:
Assay:
99%
InChI
1S/C20H16/c1-4-10-17(11-5-1)16-20(18-12-6-2-7-13-18)19-14-8-3-9-15-19/h1-16H
InChI key
MKYQPGPNVYRMHI-UHFFFAOYSA-N
SMILES string
c1ccc(cc1)\C=C(/c2ccccc2)c3ccccc3
assay
99%
mp
69-71 °C (lit.)
Quality Level
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Application
三苯乙烯是一种芳香烃，可作为起始材料，通过以氟手性锰络合物为催化剂的不对称环氧化反应制备2,2,3-三苯基环氧乙烷。也可在MnO2和NaOAc存在下，与乙酸酐反应制备二氢-4,5,5-三苯基-2(3H)-呋喃酮。
存储类别
11 - Combustible Solids
wgk
WGK 3
ppe
dust mask type N95 (US), Eyeshields, Gloves
C D van den Koedijk et al.
Biochemical pharmacology, 46(10), 1870-1872 (1993-11-17)
The binding affinity of derivatives of the triphenylethylene (TPE) antioestrogen tamoxifen and of steroidal compounds for human liver antioestrogen binding sites (AEBS) was compared with their binding affinity for rat liver AEBS. Despite the observation of some quantitative differences overall
E Bignon et al.
FEBS letters, 271(1-2), 54-58 (1990-10-01)
The activation of type I (gamma), II (beta) and III (alpha) protein kinase C (PKC) subspecies by phosphatidylserine (PS) and diacylglycerol (DAG) is inhibited by micromolar concentrations of triphenylacrylonitrile (TPE) antiestrogens. TPE A (with p-hydroxy and p-diethylaminoethoxy groups on the
Chellakkan S Blesson et al.
Steroids, 71(11-12), 993-1000 (2006-09-13)
The study was aimed to investigate the interaction of D,L-ormeloxifene (Orm), a triphenylethylene and its hydroxy derivative with estrogen receptor subtypes alpha and beta, its influence on ERE-driven transcriptional activation and progesterone receptor expression. In competitive binding experiments using human
Speculation on the mechanism of action of triphenylethylene antioestrogens.
C D van den Koedijk et al.
Biochemical pharmacology, 47(11), 1927-1937 (1994-06-01)
H Wiseman et al.
Chemico-biological interactions, 79(2), 229-243 (1991-01-01)
The azole antifungal drug ketoconazole was found to inhibit Fe(III)-ascorbate dependent lipid peroxidation using either rat liver microsomes or ox-brain phospholipid liposomes as the substrate. It also inhibited microsomal peroxidation induced by the Fe(III)-ADP/NADPH system. The related azoles, miconazole and
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