05-901R
Anti-NR2A Antibody, clone A12W, rabbit monoclonal
别名:
N-methyl D-aspartate receptor subtype 2A, N-methyl-D-aspartate receptor channel, subunit epsilon-1, N-methyl-D-aspartate receptor subunit 2A, NMDA receptor subtype 2A, glutamate receptor, ionotropic, N-methyl D-aspartate 2A
抗体产品类型
primary antibodies
表单
liquid
靶向翻译后修饰
unmodified
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储存分类代码
12 - Non Combustible Liquids
WGK
WGK 1
闪点(°F)
Not applicable
闪点(°C)
Not applicable
Han Zhao et al.
Brain and behavior, 8(7), e01004-e01004 (2018-06-02)
It is known that an interruption of acoustic input in early life will result in abnormal development of the auditory system. Here, we further show that this negative impact actually spans beyond the auditory system to the hippocampus, a system
Yukio Hattori et al.
The journal of obstetrics and gynaecology research, 43(2), 412-415 (2017-02-06)
Anti-N-methyl-d-aspartate receptor (NMDAR) limbic encephalitis is the most common form of paraneoplastic encephalitis that is associated with teratomas. Because tumor removal leads to better clinical outcomes, it is essential to reveal the location of the teratomas. This is the first
Bo Chen et al.
Journal of neuroinflammation, 19(1), 181-181 (2022-07-14)
Perioperative neurocognitive disorders (PNDs) are considered the most common postoperative complication in geriatric patients. However, its pathogenesis is not fully understood. Surgery-triggered neuroinflammation is a major contributor to the development of PNDs. Neuroinflammation can influence N-methyl-D-aspartate receptor (NMDAR) expression or
Yasunobu Murata et al.
The Journal of neuroscience : the official journal of the Society for Neuroscience, 33(11), 5040-5052 (2013-03-15)
Membrane-associated guanylate kinases (MAGUKs), including SAP102, PSD-95, PSD-93, and SAP97, are scaffolding proteins for ionotropic glutamate receptors at excitatory synapses. MAGUKs play critical roles in synaptic plasticity; however, details of signaling roles for each MAGUK remain largely unknown. Here we
Li-Hsin Chang et al.
Scientific reports, 8(1), 118-118 (2018-01-10)
Inhibition and deletion of soluble epoxide hydrolase (sEH) has been suggested to ameliorate infarction in experimental ischemic stroke possibly via vasoactive epoxyeicosatrienoic acids. However, it is unknown whether the neuroprotective mechanisms involve alteration of post-ischemic neuronal transmission and neurotrophic signaling.
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