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文件

AB9452

Sigma-Aldrich

Anti-Muscarinic Acetylcholine Receptor M2 Antibody

Chemicon®, from rabbit

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eCl@ss:
32160702
NACRES:
NA.41

生物来源

rabbit

质量水平

抗体形式

affinity purified immunoglobulin

antibody product type

primary antibodies

克隆

polyclonal

纯化方式

affinity chromatography

species reactivity

human

manufacturer/tradename

Chemicon®

technique(s)

immunohistochemistry: suitable (paraffin)

UniProt登记号

运输

dry ice

target post-translational modification

unmodified

Gene Information

human ... CHRM2(1129)

特异性

Recognizes Muscarinic Acetylcholine Receptor M2.

免疫原

Synthetic peptide from the 3rd cytoplasmic domain of human Muscarinic Acetylcholine Receptor M2.

应用

Detect Muscarinic Acetylcholine Receptor M2 using this Anti-Muscarinic Acetylcholine Receptor M2 Antibody validated for use in IH(P).
Immunohistochemistry on formalin-fixed, paraffin-embedded tissue sections.

Optimal working dilutions must be determined by the end user.
Research Category
Neuroscience
Research Sub Category
Neurotransmitters & Receptors

外形

Affinity purified immunoglobulin. Liquid in PBS, pH 7.7 with 0.01% sodium azide.

储存及稳定性

Maintain at -70°C in undiluted aliquots for up to 6 months after date of receipt. Avoid repeated freeze/thaw cycles.

其他说明

Concentration: Please refer to the Certificate of Analysis for the lot-specific concentration.

法律信息

CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany

免责声明

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

储存分类代码

10 - Combustible liquids

WGK

WGK 2

闪点(°F)

Not applicable

闪点(°C)

Not applicable


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Ítalo Martins de Oliveira et al.
Arquivos brasileiros de cardiologia, 117(5), 999-1007 (2021-08-19)
The primary factors that originate and perpetuate atrial fibrillation (AF) are electrical and anatomical substrate alterations. However, the central mechanisms governing AF perpetuation have not been elucidated yet, which is reflected on the modest results of the treatment in patients

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