ABF13
Anti-Interleukin-10 Antibody
from rabbit, purified by affinity chromatography
别名:
Interleukin-10, IL-10, Cytokine synthesis inhibitory factor, CSIF
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关于此项目
UNSPSC Code:
12352203
NACRES:
NA.41
eCl@ss:
32160702
Conjugate:
unconjugated
Clone:
polyclonal
Application:
WB
Citations:
2
biological source
rabbit
conjugate
unconjugated
antibody form
affinity isolated antibody
antibody product type
primary antibodies
clone
polyclonal
purified by
affinity chromatography
species reactivity
mouse, human
technique(s)
western blot: suitable
NCBI accession no.
UniProt accession no.
shipped in
wet ice
target post-translational modification
unmodified
Quality Level
Gene Information
human ... IL10(3586)
General description
Interleukin-10 (IL-10), originally known as Cytokine Synthesis Inhibitory Factor (CSIF) is an 18.7 kDa protein of 160 amino acid residues that shares over 80% sequence homology with the Epstein-Barr Virus protein BCRFI. IL-10 down regulates the expression of Th1 cytokines, major histocompatibility complex class II antigens, and costimulatory molecules on macrophages. It also enhaces B cell survival, proliferation, and antibody production. Knockout studies suggest that IL-10 is an immunoregulator in the intestinal tract.
~18 kDa observed.
Immunogen
GST-tagged recombinant protein corresponding to mouse Interleukin-10.
Application
Detect Interleukin-10 using this Anti-Interleukin-10 Antibody validated for use in WB.
Analysis Note
Control
Mouse Interleukin-10 recombinant protein
Mouse Interleukin-10 recombinant protein
Evaluated by Western Blot in mouse Interleukin-10 recombinant protein.
Western Blot Analysis: 0.01 µg/mL of this antibody detected Interleukin-10 on 10 µg of mouse Interleukin-10 recombinant protein.
Western Blot Analysis: 0.01 µg/mL of this antibody detected Interleukin-10 on 10 µg of mouse Interleukin-10 recombinant protein.
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存储类别
12 - Non Combustible Liquids
wgk
WGK 1
flash_point_f
Not applicable
flash_point_c
Not applicable
Exercise ameliorates high fat diet induced cardiac dysfunction by increasing interleukin 10.
Kesherwani, V; Chavali, V; Hackfort, BT; Tyagi, SC; Mishra, PK
Frontiers in Physiology null
Eva Latorre et al.
United European gastroenterology journal, 6(3), 446-453 (2018-05-19)
Inflammatory bowel diseases are consequence of an intestinal homeostasis breakdown in which innate immune dysregulation is implicated. Toll-like receptor (TLR)2 and TLR4 are immune recognition receptors expressed in the intestinal epithelium, the first physical-physiological barrier for microorganisms, to inform the
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