产品名称
Metabotropic Glutamate Receptor 5, control peptide for AB5675,
biological source
rat
form
liquid
manufacturer/tradename
Chemicon®
concentration
1 mg/mL
technique(s)
cell based assay: suitable
NCBI accession no.
UniProt accession no.
shipped in
dry ice
Quality Level
Gene Information
rat ... Grm5(24418)
Application
Antibody blocking (AB5675)
Optimal working concentration must be determined by the end user.
Optimal working concentration must be determined by the end user.
Research Category
Neuroscience
Neuroscience
Research Sub Category
Neurotransmitters & Receptors
Neurotransmitters & Receptors
Disclaimer
Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.
General description
Metabotropic Glutamate Receptor 5 (mGluR5) control peptide.
Physical form
Liquid.
Preparation Note
Store at -20°C in undiluted aliquots for up to 6 months after date of receipt. Avoid repeated freeze/thaw cycles.
Legal Information
CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany
存储类别
12 - Non Combustible Liquids
wgk
WGK 2
flash_point_f
Not applicable
flash_point_c
Not applicable
Expression of metabotropic glutamate receptor mGluR5 in human dental pulp.
Yun Sook Kim,Young Jae Kim,Sang Kyoo Paik,Yi Sul Cho,Tae Geon Kwon,Dong Kuk Ahn et al.
Journal of Endodontics null
Research design for the project information for analysis of clinical care in the Academic Medical Center, Amsterdam.
H Prins,J H Zwetsloot-Schonk,H Buller
Medinfo. MEDINFO null
A H Iyo et al.
Neuroscience, 169(4), 1567-1574 (2010-07-06)
Several lines of evidence suggest a dysfunctional glutamate system in major depressive disorder (MDD). Recently, we reported reduced levels of metabotropic glutamate receptor subtype 5 (mGluR5) in postmortem brains in MDD, however the neurobiological mechanisms that induce these abnormalities are
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