产品名称
Rabbit Anti-Sheep IgG Antibody, FITC conjugate, Chemicon®, from rabbit
biological source
rabbit
conjugate
FITC conjugate
antibody form
affinity purified immunoglobulin
antibody product type
secondary antibodies
clone
polyclonal
species reactivity
sheep
manufacturer/tradename
Chemicon®
technique(s)
immunofluorescence: suitable
shipped in
wet ice
target post-translational modification
unmodified
Quality Level
Application
DILUTION:
Optimal working dilution must be determined by the end user.
Optimal working dilution must be determined by the end user.
Rabbit anti-Sheep IgG Antibody, FITC conjugate is an antibody against Sheep IgG for use in IF.
Research Category
Secondary & Control Antibodies
Secondary & Control Antibodies
Research Sub Category
Whole Immunoglobulin Secondary Antibodies
Whole Immunoglobulin Secondary Antibodies
Biochem/physiol Actions
Sheep IgG (H&L)
Disclaimer
Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.
Immunogen
Purified Sheep IgG.
Physical form
Lyophilized from 0.02M Phosphate buffer, 0.25 M NaCl with 15 mg/mL BSA and 0.1% sodium azide.
RECONSTITUTION:
Reconstitute to 1 mg/mL with sterile distilled water.
RECONSTITUTION:
Reconstitute to 1 mg/mL with sterile distilled water.
Preparation Note
Maintain lyophilized material at 2-8°C up to one year. After reconstitution maintain at 2-8°C in an undiluted format for up to 6 months.
Legal Information
CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany
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hcodes
pcodes
Hazard Classifications
Aquatic Chronic 3
存储类别
11 - Combustible Solids
wgk
WGK 3
C Giaroni et al.
Neurogastroenterology and motility : the official journal of the European Gastrointestinal Motility Society, 25(2), e114-e126 (2013-01-03)
Intestinal ischemia and reperfusion (I/R) injury leads to abnormalities in motility, namely delay of transit, caused by damage to myenteric neurons. Alterations of the nitrergic transmission may occur in these conditions. This study investigated whether an in vitro I/R injury
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