生物来源
mouse
质量水平
偶联物
unconjugated
抗体形式
ascites fluid
抗体产品类型
primary antibodies
克隆
LH7.2, monoclonal
种属反应性
human
制造商/商品名称
Chemicon®
技术
immunocytochemistry: suitable
同位素/亚型
IgG
NCBI登记号
UniProt登记号
运输
dry ice
靶向翻译后修饰
unmodified
基因信息
human ... COL7A1(1294)
免疫原
VII型胶原。
应用
免疫组化。
最佳工作稀释度必须由最终用户进行确定。
最佳工作稀释度必须由最终用户进行确定。
抗VII型胶原抗体，C端，克隆LH7.2是用于IC的抗VII型胶原抗体。
生化/生理作用
VII型胶原蛋白的羧基末端肽。
外形
腹水。 含 0.1％ 叠氮化钠的液体。
制备说明
以方便的等分试样在 -20°C 下保存长达 12 个月。应避免反复冻/融循环。
法律信息
CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany
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储存分类代码
10 - Combustible liquids
WGK
WGK 1
闪点(°F)
Not applicable
闪点(°C)
Not applicable
Manuela Pigors et al.
Human molecular genetics, 20(9), 1811-1819 (2011-02-16)
Epidermal integrity is essential for skin functions. It is maintained by adhesive structures between keratinocytes, mainly the desmosomes and adherens junctions, which provide resistance against mechanical stress and regulate the formation of the skin barrier. As a constituent of both
Sabine E J Tanis et al.
Cell reports, 25(5), 1292-1303 (2018-11-01)
Epidermal homeostasis requires balanced progenitor cell proliferation and loss of differentiated cells from the epidermal surface. During this process, cells undergo major changes in their transcriptional programs to accommodate new cellular functions. We found that transcriptional and post-transcriptional mechanisms underlying
Distribution of basement membrane zone components in bullous lesions of subepidermal blistering diseases.
Satomi Hosoda et al.
The Journal of dermatology, 40(3), 211-212 (2013-01-09)
Cristina Chamorro et al.
Molecular therapy. Nucleic acids, 5(4), e307-e307 (2016-04-06)
Clonal gene therapy protocols based on the precise manipulation of epidermal stem cells require highly efficient gene-editing molecular tools. We have combined adeno-associated virus (AAV)-mediated delivery of donor template DNA with transcription activator-like nucleases (TALE) expressed by adenoviral vectors to
Dorothy M Supp et al.
Cell transplantation, 28(9-10), 1242-1256 (2019-07-05)
The blistering disease recessive dystrophic epidermolysis bullosa (RDEB) is caused by mutations in the gene encoding collagen VII (COL7), which forms anchoring fibrils that attach the epidermis to the dermis. Cutaneous gene therapy to restore COL7 expression in RDEB patient
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