MAB1532
Anti-Hexokinase Type I Antibody, a.a. 191-209, clone 21
ascites fluid, clone 21, Chemicon®
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关于此项目
UNSPSC Code:
12352203
NACRES:
NA.41
eCl@ss:
32160702
Conjugate:
unconjugated
Clone:
21, monoclonal
Application:
ICC, WB
Citations:
5
biological source
mouse
conjugate
unconjugated
antibody form
ascites fluid
antibody product type
primary antibodies
clone
21, monoclonal
species reactivity
bovine, rat, mouse
manufacturer/tradename
Chemicon®
technique(s)
immunocytochemistry: suitable, western blot: suitable
isotype
IgG1
shipped in
dry ice
target post-translational modification
unmodified
Quality Level
Gene Information
mouse ... Hk1(15275)
rat ... Hk1(25058)
Immunogen
Epitope: a.a. 191-209
Rat Type I hexokinase.
Application
Anti-Hexokinase Type I Antibody, a.a. 191-209, clone 21 is an antibody against Hexokinase Type I for use in IC & WB.
Research Category
Signaling
Signaling
Research Sub Category
Insulin/Energy Signaling
Kinases & Phosphatases
Insulin/Energy Signaling
Kinases & Phosphatases
Western blot: 1:500-1:2,000. Immunolocalization of Type I hexokinase: 1:500-1:2,000. Optimal working dilutions must be determined by end user.
Biochem/physiol Actions
Recognizes a segmental epitope located between residues 191-209 of the Type I hexokinase sequence.
Preparation Note
Store at -20°C in undiluted aliquots for up to 12 months. Avoid repeated freeze/thaw cycles.
Legal Information
CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany
Disclaimer
Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.
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存储类别
10 - Combustible liquids
wgk
WGK 1
flash_point_f
Not applicable
flash_point_c
Not applicable
Ning Zhang et al.
Archives of medical science : AMS, 17(2), 406-416 (2021-03-23)
Progressive accumulation of amyloid-β (Aβ) is a pathological trait of Alzheimer's disease (AD). Amyloid-β increases free radical production in neuronal cells, leading to neuronal cell death. Hormone replacement therapy can reduce the incidence of AD, and oestrogen significantly improves the
José Carlos Valle-Casuso et al.
PloS one, 7(2), e32448-e32448 (2012-03-03)
In previous work we showed that endothelin-1 (ET-1) increases the rate of glucose uptake in astrocytes, an important aspect of brain function since glucose taken up by astrocytes is used to supply the neurons with metabolic substrates. In the present
Noriko Nakamura et al.
Biology of reproduction, 79(3), 537-545 (2008-05-30)
During epididymal transit, sperm acquire the ability to initiate rapid forward progressive motility on release into the female reproductive tract or physiological media. Glycolysis is the primary source of the ATP necessary for this motility in the mouse, and several
Ana González-Sánchez et al.
Oncotarget, 7(31), 49819-49833 (2016-07-09)
Connexin43 (Cx43), the major protein forming gap junctions in astrocytes, is reduced in high-grade gliomas, where its ectopic expression exerts important effects, including the inhibition of the proto-oncogene tyrosine-protein kinase Src (c-Src). In this work we aimed to investigate the
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