产品名称
抗-淀粉样蛋白抗体,β 1-16,克隆DE2, culture supernatant, clone DE2, Chemicon®
biological source
mouse
antibody form
culture supernatant
antibody product type
primary antibodies
clone
DE2, monoclonal
species reactivity
monkey, bovine, human
manufacturer/tradename
Chemicon®
technique(s)
ELISA: suitable
immunohistochemistry: suitable (paraffin)
western blot: suitable
isotype
IgG
NCBI accession no.
UniProt accession no.
shipped in
dry ice
target post-translational modification
unmodified
Quality Level
Gene Information
human ... APP(351)
Application
抗淀粉样蛋白抗体,β1-16,克隆DE2是一种针对淀粉样蛋白的抗体,用于ELISA、WB、IH(P)。
蛋白质印迹:1:10-1:300免疫组织化学:1:10-1:200在甲醛固定、石蜡包埋的组织上,需要甲酸或微波预处理来染色阿尔兹′海默病大脑老年斑。ELISA免疫沉淀
最佳工作稀释度必须由最终用户确定。
最佳工作稀释度必须由最终用户确定。
Physical form
培养上清液。 含有0.02%叠氮化钠的液体。
Legal Information
CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany
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存储类别
10 - Combustible liquids
wgk
WGK 1
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Scientific reports, 13(1), 19828-19828 (2023-11-15)
Photobiomodulation (PBM), the process of exposing tissue to red or near-infrared light, has become a topic of great interest as a therapy for diverse pathologies, including neurodegenerative disorders. Here, we aimed to evaluate the potential beneficial effect of PBM on
Alexander J Ehrenberg et al.
Acta neuropathologica, 141(5), 651-666 (2021-03-08)
The farnesyltransferase inhibitor, Lonafarnib, reduces tau inclusions and associated atrophy in familial tauopathy models through activation of autophagy, mediated by the inhibition of farnesylation of the Ras GTPase, Rhes. While hinting at a role of Rhes in tau aggregation, it
Avindra Nath et al.
Neuromolecular medicine, 3(1), 29-39 (2003-04-01)
Studies of amyloid precursor protein transgenic mice suggest that immune responses to amyloid beta peptide (Abeta) may be instrumental in the removal of plaques from the brain, but the initial clinical trial of an Abeta vaccine in patients with Alzheimer
S100A9 knockout decreases the memory impairment and neuropathology in crossbreed mice of Tg2576 and S100A9 knockout mice model.
Kim, HJ; Chang, KA; Ha, TY; Kim, J; Ha, S; Shin, KY; Moon, C; Nacken, W; Kim, HS; Suh, YH
Testing null
Ji-Hye L Hwang et al.
Acta neuropathologica, 145(1), 1-12 (2022-12-06)
Tuberous sclerosis complex (TSC) is a neurogenetic disorder leading to epilepsy, developmental delay, and neurobehavioral dysfunction. The syndrome is caused by pathogenic variants in TSC1 (coding for hamartin) or TSC2 (coding for tuberin). Recently, we reported a progressive frontotemporal dementia-like
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