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Merck
CN

MAB5488

Anti-CRABP2 Antibody

ascites fluid, Chemicon®

别名:

CRABPII

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关于此项目

UNSPSC Code:
12352203
NACRES:
NA.41
eCl@ss:
32160702
Conjugate:
unconjugated
Clone:
monoclonal
Application:
ELISA, ICC, WB
Citations:
11
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biological source

mouse

conjugate

unconjugated

antibody form

ascites fluid

antibody product type

primary antibodies

clone

monoclonal

species reactivity

mouse, human

manufacturer/tradename

Chemicon®

technique(s)

ELISA: suitable, immunocytochemistry: suitable, western blot: suitable

isotype

IgG2a

NCBI accession no.

UniProt accession no.

shipped in

wet ice

target post-translational modification

unmodified

Quality Level

Gene Information

human ... CRABP2(1382)

Immunogen

Full length recombinant human CRABPII.

Application

Use Anti-CRABP2 Antibody (Mouse Monoclonal Antibody) validated in ELISA, WB, ICC to detect CRABP2 also known as Cellular Retinoic Acid Binding Protein II.

Biochem/physiol Actions

Reacts with Cellular Retinoic Acid Binding Protein II (CRABPII). No cross reactivity with CRABPI.

Legal Information

CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany

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存储类别

10 - Combustible liquids

wgk

WGK 1

flash_point_f

Not applicable

flash_point_c

Not applicable


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Temporal blastemal cell gene expression analysis in the kidney reveals new Wnt and related signaling pathway genes to be essential for Wilms' tumor onset.
Maschietto, M; Trape, AP; Piccoli, FS; Ricca, TI; Dias, AA; Coudry, RA; Galante et al.
Cell Death & Disease null
Shelei Pan et al.
Fluids and barriers of the CNS, 21(1), 4-4 (2024-01-09)
CSF has long been accepted to circulate throughout the subarachnoid space, which lies between the arachnoid and pia maters of the meninges. How the CSF interacts with the cellular components of the developing postnatal meninges including the dura, arachnoid, and
Shuiliang Yu et al.
Journal of experimental & clinical cancer research : CR, 41(1), 88-88 (2022-03-10)
Resistance to standard therapy is a major reason for the poor prognosis of pancreatic ductal adenocarcinoma (PDAC). Developing novel therapy to overcome PDAC drug-resistance is urgently needed. CRABP-II was highly expressed in all PDAC but not expressed in normal pancreatic
Julia Derk et al.
Developmental cell, 58(8), 635-644 (2023-03-31)
The arachnoid barrier, a component of the blood-cerebrospinal fluid barrier (B-CSFB) in the meninges, is composed of epithelial-like, tight-junction-expressing cells. Unlike other central nervous system (CNS) barriers, its' developmental mechanisms and timing are largely unknown. Here, we show that mouse
Matt J Matrongolo et al.
Development (Cambridge, England), 150(18) (2023-08-17)
Secondary lissencephaly evolved in mice due to effects on neurogenesis and the tangential distribution of neurons. Signaling pathways that help maintain lissencephaly are still poorly understood. We show that inactivating Twist1 in the primitive meninges causes cortical folding in mice.

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