产品名称
Anti-Glial Fibrillary Acidic Protein Antibody, Chemicon®, from mouse
biological source
mouse
Quality Level
antibody form
purified immunoglobulin
antibody product type
primary antibodies
clone
monoclonal
species reactivity
rat, human
manufacturer/tradename
Chemicon®
technique(s)
immunocytochemistry: suitable, western blot: suitable
isotype
IgG2a
NCBI accession no.
UniProt accession no.
shipped in
wet ice
target post-translational modification
unmodified
Gene Information
human ... GFAP(2670)
Application
Detect Glial Fibrillary Acidic Protein using this Anti-Glial Fibrillary Acidic Protein Antibody validated for use in WB & IC.
Western blot: 1:10,000 on rat brain
Optimal working dilutions must be determined by end user.
Optimal working dilutions must be determined by end user.
Physical form
Format: Purified
Purified immunoglobulin. Liquid in 0.02M phosphate buffer, 0.25 M NaCl, pH 7.6 with 0.1% sodium azide.
Analysis Note
Control
Positive: Neural tissue or astrocyte culture
Negative: Non-neural tissue or neuronal culture
Positive: Neural tissue or astrocyte culture
Negative: Non-neural tissue or neuronal culture
Other Notes
Concentration: Please refer to the Certificate of Analysis for the lot-specific concentration.
Replaces: 04-1031; 04-1062
Legal Information
CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany
存储类别
12 - Non Combustible Liquids
wgk
WGK 2
flash_point_f
Not applicable
flash_point_c
Not applicable
Takashi Ishijima et al.
Science progress, 104(4), 368504211054985-368504211054985 (2021-11-26)
By using an animal model in which inflammatory cytokines are induced in lipopolysaccharide (LPS)-injected rat brain, we investigated the induction of tumor necrosis factor alpha (TNFα), interleukin-1beta (IL-1β), and IL-6. Immunoblotting and immunohistochemistry revealed that all three cytokines were transiently
Sitthisak Thongrong et al.
Biomedical reports, 18(3), 20-20 (2023-02-18)
Chronic stress has been recognized to induce the alterations of neuronal and glial cells in the hippocampus, and is thus implicated in cognitive dysfunction. There is increasing evidence to indicate that natural compounds capable of exerting neuroprotective and antioxidant activities
Shun-Yun Cheng et al.
Molecular therapy. Nucleic acids, 35(1), 102088-102088 (2024-01-09)
Inherited retinal dystrophies caused by dominant mutations in photoreceptor (PR) cell expressed genes are a major cause of irreversible vision loss. Oligonucleotide therapy has been of interest in diseases that conventional medicine cannot target. In the early days, small interfering
全球贸易项目编号
| 货号 | GTIN |
|---|---|
| MAB5628 | 04053252727436 |