等级
SAJ first grade
蒸汽密度
2.7 (vs air)
蒸汽压
513 hPa ( 50 °C)
67.1 hPa ( 20 °C)
表单
solid
存货情况
available only in Japan
pH值（酸碱度）
7.0-8.5 (25 °C, 79.1 g/L)
储存温度
15-25°C
SMILES字符串
N.OC(O)=O
InChI
1S/CH2O3.H3N/c2-1(3)4;/h(H2,2,3,4);1H3
InChI key
ATRRKUHOCOJYRX-UHFFFAOYSA-N
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Warning
危险声明
预防措施声明
危险分类
Acute Tox. 4 Oral
储存分类代码
13 - Non Combustible Solids
WGK
WGK 1
闪点(°F)
Not applicable
闪点(°C)
Not applicable
法规信息
新产品
此项目有
Mahdokht Jouiaei et al.
Toxins, 7(3), 936-950 (2015-03-21)
Cnidarian venom research has lagged behind other toxinological fields due to technical difficulties in recovery of the complex venom from the microscopic nematocysts. Here we report a newly developed rapid, repeatable and cost effective technique of venom preparation, using ethanol
Zuzana Drobna et al.
Chemical research in toxicology, 22(10), 1713-1720 (2009-08-21)
The arsenic (+3 oxidation state) methyltransferase (As3mt) gene encodes a 43 kDa protein that catalyzes methylation of inorganic arsenic. Altered expression of AS3MT in cultured human cells controls arsenic methylation phenotypes, suggesting a critical role in arsenic metabolism. Because methylated
Letícia Cruz et al.
Journal of controlled release : official journal of the Controlled Release Society, 152(3), 370-375 (2011-03-15)
Spray-dried powders for lung delivery of sodium alendronate (SA) were prepared from hydroalcoholic solutions. Formulations display geometric particle size below to 12 μm and spherical shape associated to a hollow structure. The addition of leucine and ammonium bicarbonate leads to
Kasra Hassani et al.
PloS one, 9(4), e95007-e95007 (2014-04-17)
Protozoan parasites of Leishmania genus are able to successfully infect their host macrophage due to multiple virulence strategies that result in its deactivation. Recent studies suggest Leishmania GP63 to be a critical virulence factor in modulation of many macrophage molecules
Joachim Schessl et al.
The Journal of clinical investigation, 118(3), 904-912 (2008-02-16)
Reducing body myopathy (RBM) is a rare disorder causing progressive muscular weakness characterized by aggresome-like inclusions in the myofibrils. Identification of genes responsible for RBM by traditional genetic approaches has been impossible due to the frequently sporadic occurrence in affected
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