01-6635
氧化砷(III)
SAJ first grade, ≥99.0%
别名:
三氧化二砷, 亚砷酸
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关于此项目
经验公式（希尔记法）:
As2O3
化学文摘社编号:
分子量:
197.84
UNSPSC Code:
12352103
PubChem Substance ID:
EC Number:
215-481-4
MDL number:
InChI key
IKWTVSLWAPBBKU-UHFFFAOYSA-N
InChI
1S/As2O3/c3-1-5-2-4
SMILES string
O=[As]O[As]=O
grade
SAJ first grade
vapor pressure
0.000001 hPa ( 66 °C)
assay
≥99.0%
form
powder
availability
available only in Japan
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signalword
Danger
hcodes
Hazard Classifications
Acute Tox. 2 Oral - Aquatic Acute 1 - Aquatic Chronic 1 - Carc. 1A - Eye Dam. 1 - Skin Corr. 1B - STOT RE 1
target_organs
Respiratory system,Cardio-vascular system,Gastrointestinal tract
存储类别
6.1A - Combustible acute toxic Cat. 1 and 2 / very toxic hazardous materials
wgk
WGK 3
flash_point_f
Not applicable
flash_point_c
Not applicable
法规信息
新产品
此项目有
Yanfei Jia et al.
PloS one, 8(1), e54774-e54774 (2013-02-06)
Alpha-fetoprotein (AFP)-producing gastric cancer (AFPGC), represented by the production of AFP, has a more aggressive behavior than common gastric cancer. The underlying mechanisms are not well understood. Arsenic trioxide (As(2)O(3)) is used clinically to treat acute promyelocytic leukemia(APL) and has
Masamitsu Yanada et al.
Blood, 121(16), 3095-3102 (2013-02-16)
The optimal treatments for relapsed acute promyelocytic leukemia (APL) remain equivocal. We conducted a phase 2 study to evaluate the efficacy and feasibility of a sequential treatment consisting of induction and consolidation with arsenic trioxide (ATO), peripheral blood stem cell
Cui Li et al.
Toxicology letters, 219(3), 223-230 (2013-04-02)
Arsenic trioxide (As2O3; ATO) is clinically effective in treating acute promyelocytic leukemia (APL); however, it frequently causes cardiotoxic effects. This study was designed to investigate whether ATO could induce apoptosis of cardiac fibroblasts (CFs) that play very important roles in
Athena Kritharis et al.
Annals of hematology, 92(6), 719-730 (2013-03-16)
For more than 2,000 years, arsenic and its derivatives have shown medical utility. Owing to the toxicities and potential carcinogenicity of arsenicals, their popularity has fluctuated. The exact mechanism of action of therapeutic arsenic is not well characterized but likely
Akio Iwanami et al.
Proceedings of the National Academy of Sciences of the United States of America, 110(11), 4339-4344 (2013-02-27)
Despite their nearly universal activation of mammalian target of rapamycin (mTOR) signaling, glioblastomas (GBMs) are strikingly resistant to mTOR-targeted therapy. We analyzed GBM cell lines, patient-derived tumor cell cultures, and clinical samples from patients in phase 1 clinical trials, and
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