蒸汽压
3 mmHg ( 37 °C)
表单
liquid
存货情况
available only in Japan
浓度
0.2 M
1/5 N
密度
1 g/cm3 at 20 °C
SMILES字符串
[OH-].[Na+]
InChI
1S/Na.H2O/h;1H2/q+1;/p-1
InChI key
HEMHJVSKTPXQMS-UHFFFAOYSA-M
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警示用语：
Warning
危险声明
危险分类
Eye Irrit. 2 - Met. Corr. 1 - Skin Irrit. 2
储存分类代码
8B - Non-combustible corrosive hazardous materials
WGK
nwg
闪点(°F)
Not applicable
闪点(°C)
Not applicable
法规信息
新产品
Triggered Ca2+ influx is required for extended synaptotagmin 1-induced ER-plasma membrane tethering.
Olof Idevall-Hagren et al.
The EMBO journal, 34(17), 2291-2305 (2015-07-24)
The extended synaptotagmins (E-Syts) are ER proteins that act as Ca(2+)-regulated tethers between the ER and the plasma membrane (PM) and have a putative role in lipid transport between the two membranes. Ca(2+) regulation of their tethering function, as well
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Epigenetics, 9(11), 1461-1472 (2014-12-04)
A key step in the process of metastasis is the epithelial-to-mesenchymal transition (EMT). We hypothesized that epigenetic mechanisms play a key role in EMT and to test this hypothesis we analyzed global and gene-specific changes in DNA methylation during TGF-β-induced
Aparajita H Chourasia et al.
EMBO reports, 16(9), 1145-1163 (2015-08-02)
BNip3 is a hypoxia-inducible protein that targets mitochondria for autophagosomal degradation. We report a novel tumor suppressor role for BNip3 in a clinically relevant mouse model of mammary tumorigenesis. BNip3 delays primary mammary tumor growth and progression by preventing the
Joshua S McLane et al.
Biophysical journal, 109(2), 249-264 (2015-07-23)
Mechanical properties of the tumor microenvironment have emerged as key factors in tumor progression. It has been proposed that increased tissue stiffness can transform stromal fibroblasts into carcinoma-associated fibroblasts. However, it is unclear whether the three to five times increase
Hélène O B Gautier et al.
Nature communications, 6, 8518-8518 (2015-10-07)
Myelin regeneration can occur spontaneously in demyelinating diseases such as multiple sclerosis (MS). However, the underlying mechanisms and causes of its frequent failure remain incompletely understood. Here we show, using an in-vivo remyelination model, that demyelinated axons are electrically active
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