09180
3-氨基-4-羟基苯磺酸
technical, ≥95% (T)
别名:
2-氨基苯酚-4-磺酸
警示用语：
Warning
危险声明
危险分类
Eye Irrit. 2 - Skin Irrit. 2
储存分类代码
8A - Combustible corrosive hazardous materials
WGK
WGK 1
个人防护装备
Eyeshields, Faceshields, Gloves, type P3 (EN 143) respirator cartridges
Cyrus Daneshvar et al.
Clinical infectious diseases : an official publication of the Infectious Diseases Society of America, 49(6), 852-860 (2009-07-29)
Plasmodium knowlesi is increasingly recognized as a cause of human malaria in Southeast Asia but there are no detailed prospective clinical studies of naturally acquired infections. In a systematic study of the presentation and course of patients with acute P.
Edjair V Cabral et al.
PloS one, 7(8), e43791-e43791 (2012-08-29)
High Na(+) intake is a reality in nowadays and is frequently accompanied by renal and cardiovascular alterations. In this study, renal mechanisms underlying perinatal Na(+) overload-programmed alterations in Na(+) transporters and the renin/angiotensin system (RAS) were investigated, together with effects
Milena M Cojic et al.
Oxidative medicine and cellular longevity, 2021, 7942716-7942716 (2021-07-10)
Recent advances in vitamin D research indicate that patients with type 2 diabetes mellitus (T2DM) are suffering from vitamin D deficiency and increased oxidative stress to a variable extent, which could produce different health impacts for each individual. The novel
Patrícia Andréa da Fonseca Magalhães et al.
Life sciences, 146, 58-65 (2016-01-17)
Ischemia/reperfusion (I/R) injury and metabolic acidosis (MA) are two critical conditions that may simultaneously occur in clinical practice. The result of this combination can be harmful to the kidneys, but this issue has not been thoroughly investigated. The present study
Nicole Kemberly R Rocha et al.
Frontiers in neuroscience, 14, 579984-579984 (2020-12-19)
Neuronal-glial interactions are critical for brain homeostasis, and disruption of this process may lead to excessive glial activation and inadequate pro-inflammatory responses. Abnormalities in neuronal-glial interactions have been reported in the pathophysiology of Alzheimer's disease (AD), where lithium has been
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