36890
1,2-二氟苯
purum, ≥96.0% (GC)
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经验公式（希尔记法）:
C6H4F2
化学文摘社编号:
分子量:
114.09
EC Number:
206-680-7
UNSPSC Code:
12352100
PubChem Substance ID:
Beilstein/REAXYS Number:
1905113
MDL number:
grade
purum
assay
≥96.0% (GC)
refractive index
n20/D 1.443 (lit.), n20/D 1.445
bp
92 °C (lit.)
mp
−34 °C (lit.)
density
1.158 g/mL at 25 °C (lit.)
SMILES string
Fc1ccccc1F
InChI
1S/C6H4F2/c7-5-3-1-2-4-6(5)8/h1-4H
InChI key
GOYDNIKZWGIXJT-UHFFFAOYSA-N
法规信息
新产品
此项目有
I M Rietjens et al.
Biochemistry, 32(18), 4801-4812 (1993-05-11)
In the present study, a hypothesis is presented for the prediction of the regioselectivity of cytochrome P-450 catalyzed hydroxylation of fluorobenzenes. The regioselectivity of the in vivo hydroxylation of fluorobenzene, 1,2-difluorobenzene, 1,3-difluorobenzene, 1,2,3-triluorobenzene, and 1,2,4-triflurobenzene could be predicted within 6%
Aujin Kim et al.
The Journal of organic chemistry, 71(5), 2170-2172 (2006-02-25)
A short, high-yielding synthesis of differentially substituted resorcinol derivatives has been developed that utilizes 1,3-difluorobenzene as the starting material and employs sequential nucleophilic aromatic substitution (S(N)Ar) reactions to generate desymmetrized products. The scope and limitations of the second S(N)Ar reaction
Aubrey Yao et al.
Anesthesia and analgesia, 106(6), 1759-1764 (2008-05-24)
Gamma-aminobutyric acid type A receptor potentiation and/or N-methyl-d-aspartate (NMDA) receptor inhibition might explain the anesthetic properties of fluorinated aromatic compounds. We hypothesized that depression of dorsal horn neuronal responses to noxious stimulation would correlate with the magnitude of effect of
Joseph F Antognini et al.
Anesthesia and analgesia, 104(4), 822-828 (2007-03-23)
Previous work demonstrated that isoflurane and halothane act on the spinal cord rather than on the brain to produce immobility in the face of noxious stimulation. These anesthetics share many effects on specific receptors, and thus do not test the
Takafumi Horishita et al.
Anesthesia and analgesia, 107(5), 1579-1586 (2008-10-22)
Many inhaled anesthetics inhibit voltage-gated sodium channels at clinically relevant concentrations, and suppression of neurotransmitter release by these anesthetics results, at least partly, from decreased presynaptic sodium channel activity. Volatile aromatic anesthetics can inhibit N-methyl-D-aspartate (NMDA) receptor function and enhance
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