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Merck
CN

57579

Supelco

L-胱氨酸 盐酸盐 溶液

10 mM amino acid in 0.1 M HCl, analytical standard

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About This Item

经验公式(希尔记法):
C6H12N2O4S2 · HCl
CAS号:
分子量:
276.76
Beilstein:
4828867
MDL编号:
UNSPSC代码:
85151701
PubChem化学物质编号:
NACRES:
NA.24

等级

analytical standard

质量水平

表单

liquid

分析物化学类别

amino acids, peptides, proteins

浓度

10 mM amino acid in 0.1 M HCl

技术

HPLC: suitable
gas chromatography (GC): suitable

颜色

colorless

应用

food and beverages

包装形式

single component solution

储存温度

2-8°C

SMILES字符串

Cl.N[C@@H](CSSC[C@H](N)C(O)=O)C(O)=O

InChI

1S/C6H12N2O4S2.ClH/c7-3(5(9)10)1-13-14-2-4(8)6(11)12;/h3-4H,1-2,7-8H2,(H,9,10)(H,11,12);1H/t3-,4-;/m0./s1

InChI key

IOCJWNPYGRVHLN-MMALYQPHSA-N

应用

有关合适的仪器技术的更多信息,请参阅产品′的检验报告。想要获得更多支持,请联系技术服务部。

象形图

Corrosion

警示用语:

Warning

危险声明

预防措施声明

危险分类

Met. Corr. 1

储存分类代码

8B - Non-combustible corrosive hazardous materials

WGK

nwg

闪点(°F)

Not applicable

闪点(°C)

Not applicable

个人防护装备

Faceshields, Gloves, Goggles


历史批次信息供参考:

分析证书(COA)

Lot/Batch Number

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访问文档库

Ganeswara Rao Musada et al.
Developmental neurobiology, 80(3-4), 98-110 (2020-04-09)
Muller glia are the predominant glial cell type in the retina, and they structurally and metabolically support retinal neurons. Wnt/β-catenin signaling pathways play essential roles in the central nervous system, including glial and neuronal differentiation, axonal growth, and neuronal regeneration.
Halina T Norbertczak et al.
Tissue engineering. Part C, Methods, 26(11), 565-576 (2020-10-15)
Intervertebral disc (IVD) degeneration is a major cause of back pain. Current surgical interventions have limitations. An alternative approach is to replace degenerated IVDs with a natural biological scaffold. The removal of cellular components from human IVDs should render them
Margaret E Torrence et al.
eLife, 10 (2021-03-02)
The mechanistic target of rapamycin complex 1 (mTORC1) stimulates a coordinated anabolic program in response to growth-promoting signals. Paradoxically, recent studies indicate that mTORC1 can activate the transcription factor ATF4 through mechanisms distinct from its canonical induction by the integrated

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