AV39521
抗-TEAD1 (AB1) 兔抗
affinity isolated antibody
别名:
抗-TEA结构域家族成员1（SV40转录增强子因子）
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关于此项目
NACRES:
NA.41
UNSPSC Code:
12352203
Conjugate:
unconjugated
Clone:
polyclonal
Application:
immunohistochemistry
western blot
western blot
Species reactivity:
horse, bovine, rabbit, human, rat, guinea pig, mouse, dog
Citations:
3
Technique(s):
immunohistochemistry: suitable
western blot: suitable
western blot: suitable
Uniprot accession no.:
产品名称
抗-TEAD1 (AB1) 兔抗, affinity isolated antibody
biological source
rabbit
conjugate
unconjugated
antibody form
affinity isolated antibody
antibody product type
primary antibodies
clone
polyclonal
form
buffered aqueous solution
mol wt
46 kDa
species reactivity
horse, bovine, rabbit, human, rat, guinea pig, mouse, dog
concentration
0.5 mg - 1 mg/mL
technique(s)
immunohistochemistry: suitable
western blot: suitable
NCBI accession no.
UniProt accession no.
shipped in
wet ice
storage temp.
−20°C
target post-translational modification
unmodified
Quality Level
Gene Information
human ... TEAD1(7003)
Application
兔抗-TEAD1抗体适用于以0.25 μg/ml的浓度用于western blot应用，以16 μg/ml浓度进行免疫组化分析(IHC)。
Biochem/physiol Actions
TEAD1是一种转录增强子。可与骨骼肌特异性辅因子相互作用，促进骨骼肌基因表达。TEAD1基因突变是Sveinsson脉络膜视网膜萎缩的致病因。
Disclaimer
除非我们的产品目录或产品附带的其他公司文档另有说明，否则我们的产品仅供研究使用，不得用于任何其他目的，包括但不限于未经授权的商业用途、体外诊断用途、离体或体内治疗用途或任何类型的人类或动物食用或应用。
General description
TEAD1是一种转录因子，可阻断人子宫蜕膜细胞中催乳素的表达。TEAD1突变与Sveinsson脉络膜视网膜萎缩有关。′
兔抗TEAD1抗体可识别猪、斑马鱼、人、小鼠、大鼠、牛和犬TEAD1。
兔抗TEAD1抗体可识别猪、斑马鱼、人、小鼠、大鼠、牛和犬TEAD1。
Immunogen
对应人TEAD1 C末端区域的合成肽
Other Notes
合成肽位于以下区域内： YMMNSVLENFTILLVVTNRDTQETLLCMACVFEVSNSEHGAQHHIYRLVK
Physical form
本品为溶于1x PBS缓冲液的纯化抗体，含 0.09% (w/v) 叠氮化钠和2%蔗糖。
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存储类别
10 - Combustible liquids
wgk
WGK 1
flash_point_f
Not applicable
flash_point_c
Not applicable
法规信息
常规特殊物品
此项目有
Ragnheidur Fossdal et al.
Human molecular genetics, 13(9), 975-981 (2004-03-16)
Sveinsson's chorioretinal atrophy (SCRA), also referred to as helicoid peripapillary chorioretinal degeneration or atrophia areata, is an autosomal dominant eye disease, characterized by symmetrical lesions radiating from the optic disc involving the retina and the choroid. Genome-wide linkage analysis mapped
Cherie A Kessler et al.
Molecular and cellular endocrinology, 295(1-2), 32-38 (2008-09-09)
Forced overexpression of TEAD1 in human uterine fibroblast (HUF) and human endometrial stromal cells markedly inhibited prolactin promoter activity in both cell types in a dose-dependent manner, with maximal inhibition of greater than 90%. Conversely, the knockdown of TEAD1 expression
Shohei Ikeda et al.
JACC. Basic to translational science, 4(5), 611-622 (2019-11-27)
Patients with diabetes are more prone to developing heart failure in the presence of high blood pressure than those without diabetes. Yes-associated protein (YAP), a key effector of the Hippo signaling pathway, is persistently activated in diabetic hearts, and YAP
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