产品名称
cGMP 酶免疫检测试剂盒, sufficient for 96 assays
usage
sufficient for 96 assays
technique(s)
ELISA: suitable
shipped in
wet ice
storage temp.
−20°C
Quality Level
Gene Information
human ... PRKG1(5592)
Application
cGMP 酶免疫测定试剂盒已用于定量检测寄生虫组织和小鼠主动脉匀浆中的环磷酸鸟苷（cGMP）浓度。它也被用于评估鸟苷酸环化酶的活性。
非放射性竞争免疫测定法，用于定量cGMP，适用于生物流体（血清、血浆、尿液和唾液）、组织培养基样品或环状核苷酸浓度极低的样品。 本试剂盒采用 cGMP 多克隆抗体竞争性结合 cGMP 或已与碱性磷酸酶分子共价连接的 cGMP。 本测定在涂有抗兔IgG抗体的96孔板上进行。 向孔中添加底物产生有色终产物后，用多孔板检测仪在405 nm处读取结果。颜色的强度与孔中存在的cGMP浓度成反比。
General description
cGMP 酶免疫测定试剂盒是一种非放射性竞争免疫测定，用于生物体液（血清、血浆、尿液和唾液）、组织培养基样品或环核苷酸浓度极低的样品中的环磷酸鸟苷（cGMP）的定量检测。本试剂盒采用 cGMP 多克隆抗体竞争性结合 cGMP 或已与碱性磷酸酶分子共价连接的 cGMP。本免疫测定在涂有抗兔IgG抗体的96孔板上进行。向孔中添加底物产生有色终产物后，用多孔板检测仪在405 nm处读取结果。颜色的强度与孔中存在的cGMP浓度成反比。
signalword
Danger
Hazard Classifications
Acute Tox. 2 Inhalation - Acute Tox. 3 Dermal - Acute Tox. 4 Oral - Eye Dam. 1 - Flam. Liq. 2 - Skin Corr. 1A - STOT SE 3
target_organs
Respiratory system
存储类别
3 - Flammable liquids
flash_point_f
12.2 °F - closed cup
flash_point_c
-11 °C - closed cup
法规信息
危险化学品
低风险生物材料
常规特殊物品
易制毒化学品（2类）
此项目有
Identification of a novel Arabidopsis thaliana nitric oxide-binding molecule with guanylate cyclase activity in vitro
Mulaudzi T, et al.
Febs Letters, 585(17), 2693-2697 (2011)
Inhibition of MPO (Myeloperoxidase) Attenuates Endothelial Dysfunction in Mouse Models of Vascular Inflammation and Atherosclerosis
Cheng D, et al.
Arteriosclerosis, Thrombosis, and Vascular Biology, 39(7), 1448-1457 (2019)
Genistein from Flemingia vestita (Fabaceae) enhances NO and its mediator (cGMP) production in a cestode parasite, Raillietina echinobothrida
Das B, et al.
Parasitoloty, 134(10), 1457-1463 (2007)
Sara Falvo et al.
Animals : an open access journal from MDPI, 11(6) (2021-07-03)
The quail Coturnix coturnix is a seasonal breeding species, with the annual reproductive cycle of its testes comprising an activation phase and a regression phase. Our previous results have proven that the testicular levels of both 17β-estradiol (E2) and androgens
David Cheng et al.
Arteriosclerosis, thrombosis, and vascular biology, 39(7), 1448-1457 (2019-05-03)
Objective- Inflammation-driven endothelial dysfunction initiates and contributes to the progression of atherosclerosis, and MPO (myeloperoxidase) has been implicated as a potential culprit. On release by circulating phagocytes, MPO is thought to contribute to endothelial dysfunction by limiting NO bioavailability via
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