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Merck
CN

EHU020381

MISSION® esiRNA

targeting human CADM4

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NACRES:
NA.51
UNSPSC Code:
41105324
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description

Powered by Eupheria Biotech

product line

MISSION®

form

lyophilized powder

esiRNA cDNA target sequence

CGGATAACGGCACCTACACTTGCGAGGCGTCCAATAAGCACGGCCATGCGAGGGCGCTCTACGTACTTGTGGTCTACGACCCTGGTGCGGTGGTAGAGGCTCAGACGTCGGTTCCCTATGCCATTGTGGGCGGCATCCTGGCGCTGCTGGTGTTTCTGATCATATGTGTGCTAGTGGGCATGGTCTGGTGCTCGGTACGGCAGAAGGGTTCCTATCTGACCCACGAAGCCAGTGGCTTGGATGAACAGGGAGAAGCAAGAGAAGCCTTCCTCAATGGCAGCGACGGACACAAGAGGAAAGAGGAATTCTTCATCTGACCCTATCCCCACCCCAGGCCTAGGCCTGGGCCTGGGCTGGGGTCCCCCCCACTGCCAGCTGCAAGGAACCAGCAAAGACATTTACCAGAGTCTGGGATGGTGGGCTTCTCCCCCCACCACTAACACCTCAGACGCTTGGGCAGGGATGGGGGTGTTGGATGCCTGGATCTCTGTAAGGGCCAG

Ensembl | human accession no.

NCBI accession no.

shipped in

ambient

storage temp.

−20°C

Quality Level

General description

MISSION® esiRNA are endoribonuclease prepared siRNA. They are a heterogeneous mixture of siRNA that all target the same mRNA sequence. These multiple silencing triggers lead to highly-specific and effective gene silencing.

For additional details as well as to view all available esiRNA options, please visit SigmaAldrich.com/esiRNA.

Legal Information

MISSION is a registered trademark of Merck KGaA, Darmstadt, Germany

存储类别

10 - Combustible liquids

flash_point_f

Not applicable

flash_point_c

Not applicable

法规信息

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历史批次信息供参考:

分析证书(COA)

Lot/Batch Number

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Fang Luo et al.
The international journal of biochemistry & cell biology, 123, 105750-105750 (2020-04-24)
Cell adhesion molecule 4 (CADM4) is downregulated in many human cancers. However, CADM4 expression levels in human non-small cell lung cancer (NSCLC) tissues and its roles in NSCLC progression remain unknown. Our study aims to address these issues. We examined
Shruthy Suresh et al.
PLoS genetics, 13(3), e1006650-e1006650 (2017-03-09)
Hepatocellular carcinoma (HCC) is the fifth most common solid tumor in the world and the third leading cause of cancer-associated deaths. A Sleeping Beauty-mediated transposon mutagenesis screen previously identified mutations that cooperate with MYC to accelerate liver tumorigenesis. This revealed

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