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Merck
CN

EHU090031

MISSION® esiRNA

targeting human MAPK8IP3

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NACRES:
NA.51
UNSPSC Code:
41105324

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产品名称

MISSION® esiRNA, targeting human MAPK8IP3

description

Powered by Eupheria Biotech

product line

MISSION®

form

lyophilized powder

esiRNA cDNA target sequence

TTCCAGGGAGCAACAGAGAGGCCACCAAGCAGAGGCCCGTGGGGCTGAGGATGGAGCCGCCCCCAGCCGACTCCAAGCCCGCAGAGGGCAGACGCCACCCTGGACTGCTCTCCCTGCCCAGCTGGGCCTCTCTGGCCTATTCCTACCTTCCAGGCCCACTGCACTCCTGTCTGGGAGGCCCTTATGAGGGCAGCCCAGCCCCCGCACCCACCCCCAACCAGAGAAGCACAGATCTTGGGGAGCTGCCCCACAAGCCCCGCTGGCCACCGAGGGCTGCAGCCGCTGCGCTGCCGGCTTCTCCCCACCACCCTGCCACCTCCACTGTGATGTATGTCCGCTCCCTCGTCTGTTCCCCCAGGATCTCGAAGTGACTCCGGGCTGAGCAGTGGGGCGGCTGGGGGAGGGGTGACGATTCTCCTCAGGCTTTG

Ensembl | human accession no.

ENSG00000138834

NCBI accession no.

NM_015133

shipped in

ambient

storage temp.

−20°C

Quality Level

100

Gene Information

human ... MAPK8IP3(23162), MAPK8IP3(23162)

General description

MISSION® esiRNA are endoribonuclease prepared siRNA. They are a heterogeneous mixture of siRNA that all target the same mRNA sequence. These multiple silencing triggers lead to highly-specific and effective gene silencing.

For additional details as well as to view all available esiRNA options, please visit SigmaAldrich.com/esiRNA.

Legal Information

MISSION is a registered trademark of Merck KGaA, Darmstadt, Germany

存储类别

10 - Combustible liquids

flash_point_f

Not applicable

flash_point_c

Not applicable

法规信息

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Hao Guo et al.
International immunopharmacology, 81, 106272-106272 (2020-02-18)
Rheumatoid arthritis (RA) is a chronic inflammatory autoimmune disease. However, the pathogenesis of RA is not fully understood. Here, we reported that c-Jun NH2-terminal kinase (JNK)/stress-activated protein kinase-associated protein 1 (JSAP1, also known as JNK-interacting protein 3 (JIP3)) was significantly
Peiyuan Zhang et al.
Nature communications, 11(1), 2487-2487 (2020-05-20)
Cancer stem-like cells (CSCs) are the tumorigenic cell subpopulation and contribute to cancer recurrence and metastasis. However, the understanding of CSC regulatory mechanisms remains incomplete. By transcriptomic analysis, we identify a scaffold protein SH3RF3 (also named POSH2) that is upregulated

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