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Merck
CN

EHU150651

MISSION® esiRNA

targeting human HLX

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NACRES:
NA.51
UNSPSC Code:
41105324

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产品名称

MISSION® esiRNA, targeting human HLX

description

Powered by Eupheria Biotech

product line

MISSION®

form

lyophilized powder

esiRNA cDNA target sequence

ACAACAACAGCAGCAGCAACAGCCGCAGCAGCAACAGCCTCCGCCTCCGCCCCGGGCTGGCGCCCTGCAGCCCCCGGCCTCGGGGACGCGAGTGGTTCCGAACCCCCACCACAGTGGCTCTGCCCCGGCCCCCTCCAGCAAAGACCTCAAATTTGGAATTGACCGCATTTTATCTGCAGAATTTGACCCAAAAGTCAAAGAAGGCAACACGCTGAGAGATCTCACTTCCCTGCTAACCGGTGGGCGGCCCGCCGGGGTGCACCTCTCAGGCCTGCAGCCCTCGGCCGGCCAGTTCTTCGCATCTCTAGATCCCATTAACGAGGCTTCTGCAATCCTGAGTCCCTTAAACTCGAACCCAAGAAATTCAGTTCAGCATCAGTTCCAAGACACGTTTCCAGGTCCCTATGCTGTGCTCACGAAGGACACCATGCCGCAGACGTACAAAAGGAAGCGTTCATGGT

Ensembl | human accession no.

ENSG00000136630

NCBI accession no.

NM_021958

shipped in

ambient

storage temp.

−20°C

Quality Level

100

Gene Information

human ... HLX(3142), HLX(3142)

General description

MISSION esiRNA are endoribonuclease prepared siRNA. They are a heterogeneous mixture of siRNA that all target the same mRNA sequence. These multiple silencing triggers lead to highly-specific and effective gene silencing.

For additional details as well as to view all available esiRNA options, please visit SigmaAldrich.com/esiRNA.

Legal Information

MISSION is a registered trademark of Merck KGaA, Darmstadt, Germany

存储类别

10 - Combustible liquids

flash_point_f

Not applicable

flash_point_c

Not applicable

法规信息

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Xia-Yin Zhu et al.
Oncology letters, 20(2), 1888-1896 (2020-07-30)
Acute myelogenous leukemia (AML) is a class of malignant tumors derived from hematopoietic stem or progenitor cells. The H2.0-like homeobox gene (HLX) encodes transcription factors that function in promoting normal hematopoietic cell proliferation and tumor immunity. The present study analyzed
Jixin Zhong et al.
Journal of autoimmunity, 53, 95-104 (2014-06-18)
Unlike genetic alterations, epigenetic modifications are reversible and amenable to pharmacological interventions, which make them appealing targets for clinical therapy. However, little is known about epigenetic regulation in experimental autoimmune encephalomyelitis (EAE). Here we demonstrated that methyl-CpG-binding domain protein 2

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