Merck
CN

L5254

Sigma-Aldrich

1-棕榈酰-sn-甘油-3-磷酸胆碱

synthetic, ≥99%

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别名:
PC(16:0/0:0), 2-1十六烷酰基-sn-甘油-3-磷酸胆碱, 3-sn-溶血磷脂酰胆碱,1-棕榈酰, L-α-棕榈酰溶血磷脂酰胆碱, L-γ-棕榈酰-α-溶血卵磷脂, 溶血卵磷脂,棕榈酰
经验公式(希尔记法):
C24H50NO7P
CAS号:
分子量:
495.63
Beilstein:
5385682
MDL编号:
PubChem化学物质编号:
NACRES:
NA.25

生物来源

synthetic

质量水平

检测方案

≥99%

形式

powder

官能团

phospholipid

脂质类型

phosphoglycerides

运输

ambient

储存温度

−20°C

SMILES字符串

O[C@](COP([O-])(OCC[N+](C)(C)C)=O)([H])COC(CCCCCCCCCCCCCCC)=O

InChI

1S/C24H50NO7P/c1-5-6-7-8-9-10-11-12-13-14-15-16-17-18-24(27)30-21-23(26)22-32-33(28,29)31-20-19-25(2,3)4/h23,26H,5-22H2,1-4H3/t23-/m1/s1

InChI key

ASWBNKHCZGQVJV-HSZRJFAPSA-N

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相关类别

生化/生理作用

1-棕榈酰-sn-甘油-3-磷酸胆碱可用作底物以鉴定、区分和表征溶血磷酸胆碱转酰基转移酶。

储存分类代码

11 - Combustible Solids

WGK

WGK 3

闪点(°F)

Not applicable

闪点(°C)

Not applicable

个人防护装备

Eyeshields, Gloves, type N95 (US)


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Inna A Nikonorova et al.
Nucleic acids research, 42(20), 12779-12788 (2014-10-19)
TRPM7 is an essential and ubiquitous channel-kinase regulating cellular influx of Mg2+. Although TRPM7 mRNA is highly abundant, very small amount of the protein is detected in cells, suggesting post-transcriptional regulation of trpm7 gene expression. We found that TRPM7 mRNA
Akira Sato et al.
Chemical biology & drug design, 80(3), 417-425 (2012-05-18)
Lysophosphatidylcholine, a major phospholipid component of oxidized low-density lipoprotein, is implicated in many inflammatory diseases, including atherosclerosis. We previously reported that Asp-hemolysin-related synthetic peptide (P21) composed of 21 amino acid residues markedly inhibits the bioactivities of oxidized low-density lipoprotein and
Lu-Cheng Cao et al.
Kidney international, 66(5), 1890-1900 (2004-10-22)
In cultured renal epithelial cells, exposure to oxalate, a constituent of many kidney stones, elicits a cascade of responses that often leads to cell death. Oxalate toxicity is mediated via generation of reactive oxygen species (ROS) in a process that
J Wang et al.
Circulation, 104(22), 2645-2648 (2001-11-28)
Lysophosphatidylcholine (LPC), a naturally occurring phospholipid metabolite, accumulates in the ischemic heart and causes extracellular K(+) accumulation and action potential shortening. LPC has been incriminated as a biochemical trigger of lethal cardiac arrhythmias, but the underlying mechanisms remain poorly understood.
Thomas Pulinilkunnil et al.
Cardiovascular research, 59(3), 788-797 (2003-09-23)
Lipoprotein lipase (LPL) mediated hydrolysis of circulating triglyceride (TG)-rich lipoproteins provides the heart with fatty acids. The present study was designed to investigate the influence of circulating TG and their lipolysis in facilitating translocation of LPL from the underlying cardiomyocyte

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