PLA0092
Rabbit anti-MAD1 Antibody, Affinity Purified
Powered by Bethyl Laboratories, Inc.
别名:
HsMAD1, MAD1, MAD1 (mitotic arrest deficient; yeast; homolog)-like 1, MAD1 mitotic arrest deficient-like 1, MAD1-like protein 1, PIG9, TP53I9, TXBP181, hMAD1, homolog-like 1, mitotic arrest deficient 1-like protein 1, mitotic checkpoint MAD1 protein homolog, mitotic-arrest deficient 1, tax-binding protein 181, tumor protein p53 inducible protein 9, yeast
生物来源
rabbit
质量水平
偶联物
unconjugated
抗体形式
affinity purified immunoglobulin
抗体产品类型
primary antibodies
克隆
polyclonal
种属反应性
human
浓度
1 mg/mL
技术
immunohistochemistry: 1:500- 1:2,000
immunoprecipitation (IP): 1-4 μg/mg
western blot: 1:5,000- 1:15,000
登记号
Q9Y6D9
运输
wet ice
储存温度
2-8°C
靶向翻译后修饰
unmodified
基因信息
rabbit ... MAD1(8379)
免疫原
The epitope recognized by PLA0092 maps to a region between residues 1 and 50 of human Mitotic arrest deficient-like 1 using the numbering given in Swiss-Prot entry Q9Y6D9 (GeneID 8379).
外形
Tris-citrate/phosphate buffer, pH 7 to 8 containing 0.09% Sodium Azide
其他说明
MAD1 (Mitotic Arrest Deficiency 1) is a component of the mitotic spindle-assembly checkpoint that ensures that all chromosomes are aligned at the metaphase plate before the onset of anaphase. MAD1 functions as a homodimer that interacts with and regulates MAD2. MAD1 may play a role in tumor suppression.
免责声明
Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.
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储存分类代码
12 - Non Combustible Liquids
WGK
nwg
闪点(°F)
Not applicable
闪点(°C)
Not applicable
法规信息
新产品
此项目有
Hirohisa Kyogoku et al.
Developmental cell, 41(3), 287-298 (2017-05-10)
Chromosome segregation during meiosis in oocytes is error prone. The uniquely large cytoplasmic size of oocytes, which provides support for embryogenesis after fertilization, might be a predisposing factor for meiotic errors. However, this hypothesis remains unproven. Here, we show that
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