RAB0073
人MIP-1α / CCL3 ELISA试剂盒
for serum, plasma, cell culture supernatant, urine
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关于此项目
NACRES:
NA.32
UNSPSC Code:
41116158
Gene information:
human ... CCL3(6348)
Input:
sample type serum
sample type urine
sample type cell culture supernatant(s)
sample type plasma
sample type urine
sample type cell culture supernatant(s)
sample type plasma
Species reactivity:
human
Storage temp.:
−20°C
Shipped in:
wet ice
species reactivity
human
packaging
kit of 96 wells (12 strips x 8 wells)
technique(s)
ELISA: suitable, capture ELISA: suitable
input
sample type serum
sample type urine
sample type cell culture supernatant(s)
sample type plasma
assay range
inter-assay cv: <12%
intra-assay cv: <10%
sensitivity: 6 pg/mL
standard curve range: 4.12-3000 pg/mL
detection method
colorimetric
shipped in
wet ice
storage temp.
−20°C
Gene Information
human ... CCL3(6348)
Application
请参考Protocol了解详情。
General description
The Human MIP-1 α (Macrophage Inflammatory Protein-1 α) ELISA (Enzyme-Linked Immunosorbent Assay) kit is an in vitro enzyme-linked immunosorbent assay for the quantitative measurement of human MIP-1 α in serum, plasma, cell culture supernatants and urine.
Immunogen
Recombinant Human MIP-1 α
Other Notes
A sample Certificate of Analysis is available for this product.
Please type the word sample in the text box provided for lot number.
Please type the word sample in the text box provided for lot number.
signalword
Warning
hcodes
pcodes
Hazard Classifications
Met. Corr. 1
存储类别
8B - Non-combustible corrosive hazardous materials
flash_point_f
Not applicable
flash_point_c
Not applicable
法规信息
新产品
此项目有
Gakwaya Habiyaremye et al.
Fluids and barriers of the CNS, 14(1), 35-35 (2017-12-13)
Neuroinflammation has been implicated in the pathophysiology of post-hemorrhagic hydrocephalus (PHH) of prematurity, but no comprehensive analysis of signaling molecules has been performed using human cerebrospinal fluid (CSF). Lumbar CSF levels of key cytokines (IL-1α, IL-1β, IL-4, IL-6, IL-8, IL-10
Manu N Capoor et al.
International journal of molecular sciences, 22(5) (2021-03-04)
Previously, we proposed the hypothesis that similarities in the inflammatory response observed in acne vulgaris and degenerative disc disease (DDD), especially the central role of interleukin (IL)-1β, may be further evidence of the role of the anaerobic bacterium Cutibacterium (previously
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