S8067
Anti-phospho-Synapsin I (pSer9) antibody produced in rabbit
~1 mg/mL, affinity isolated antibody, buffered aqueous solution
产品名称
Anti-phospho-Synapsin I (pSer9) antibody produced in rabbit, ~1 mg/mL, affinity isolated antibody, buffered aqueous solution
biological source
rabbit
conjugate
unconjugated
antibody form
affinity isolated antibody
antibody product type
primary antibodies
clone
polyclonal
form
buffered aqueous solution
species reactivity
Xenopus, human, chicken, rat, mouse
concentration
~1 mg/mL
technique(s)
immunohistochemistry: 1:1,000
indirect immunofluorescence: 1:1,000
western blot: 1:1,000 using rat brain hipoccampal tissue homogenate
UniProt accession no.
shipped in
dry ice
storage temp.
−20°C
target post-translational modification
phosphorylation (pSer9)
Quality Level
Gene Information
human ... SYN1(6853)
mouse ... Syn1(20964)
rat ... Syn1(24949)
Disclaimer
Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.
Immunogen
synthetic phosphorylated peptide derived from the region of rat synapsin 1 that is phosphorylated on serine 9.
Physical form
Solution in 10 mM HEPES buffer, pH 7.5, and 150 mM NaCl containing 100 μg/ml bovine serum albumin and 50% glycerol.
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存储类别
10 - Combustible liquids
wgk
WGK 1
flash_point_f
Not applicable
flash_point_c
Not applicable
ppe
Eyeshields, Gloves, multi-purpose combination respirator cartridge (US)
法规信息
常规特殊物品
此项目有
Fang Du et al.
Aging cell, 20(5), e13368-e13368 (2021-05-06)
Mitochondrial dysfunction is one of the early pathological features of Alzheimer's disease (AD). Accumulation of cerebral and mitochondrial Aβ links to mitochondrial and synaptic toxicity. We have previously demonstrated the mechanism by which presequence peptidase (PITRM1)-mediated clearance of mitochondrial Aβ
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