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SAB1305552

Anti-MAP1LC3A antibody

Name: Anti-MAP1LC3A antibody
Brand: SIGMA

mouse monoclonal, 166AT1234

别名:

MAP1 轻链 3 样蛋白 1, MAP1LC3A, 微管相关蛋白 1A/1B 轻链 3A, 自噬相关泛素样修饰物 LC3 A, 自噬相关蛋白 LC3 A

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关于此项目

UNSPSC Code:

12352203

NACRES:

NA.41

Clone:

166AT1234, monoclonal

Species reactivity:

rat, mouse, human

Application:

IF, IHC, WB

Citations:

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属性

产品名称

单克隆抗-LC3（APG8）小鼠抗, clone 166AT1234, IgG fraction of antiserum, buffered aqueous solution

Quality Level

100

biological source

mouse

antibody form

IgG fraction of antiserum

antibody product type

primary antibodies

clone

166AT1234, monoclonal

form

buffered aqueous solution

mol wt

14272 Da

species reactivity

rat, mouse, human

technique(s)

immunofluorescence: 1:25, immunohistochemistry: 1:50-1:100, western blot: 1:1000

isotype

IgG1κ

NCBI accession no.

NP_115903.1

UniProt accession no.

Q9H492

shipped in

wet ice

storage temp.

−20°C

target post-translational modification

unmodified

Gene Information

human ... MAP1LC3A(84557)

说明

Physical form

以含有 0.09％（W/V）叠氮化钠的 PBS 形式提供

Disclaimer

除非我们的产品目录或产品附带的其他公司文档另有说明，否则我们的产品仅供研究使用，不得用于任何其他目的，包括但不限于未经授权的商业用途、体外诊断用途、离体或体内治疗用途或任何类型的消费或应用于人类或动物。

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存储类别

10 - Combustible liquids

flash_point_f

Not applicable

flash_point_c

Not applicable

法规信息

常规特殊物品

低风险生物材料

常规特殊物品

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历史批次信息供参考:

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Lot/Batch Number

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Potential role of autophagy in T‑cell survival in polymyositis and dermatomyositis.

Xiaoming Shu et al.

Molecular medicine reports, 16(2), 1180-1188 (2017-06-07)

Peripheral blood T lymphocytopenia has previously been identified in polymyositis/dermatomyositis (PM/DM) patients. Therefore, the present study aimed to examine the potential role of autophagy in peripheral blood T cell survival in PM/DM patients. Transmission electron microscopy was used to detect

NLRP3 Inflammasome Deficiency Alleviates Inflammation and Oxidative Stress by Promoting PINK1/Parkin-Mediated Mitophagy in Allergic Rhinitis Mice and Nasal Epithelial Cells.

Hong Ding et al.

Journal of asthma and allergy, 17, 717-731 (2024-08-06)

Accumulating evidence indicates that oxidative stress and inflammation are the pathological basis of allergic diseases. Inhibition of NOD-like receptor family pyrin domain-containing 3 (NLRP3) inflammasome could ameliorate allergic rhinitis (AR). Here, we explored the effects and mechanisms that underlie NLRP3

p53 Deacetylation Alleviates Sepsis-Induced Acute Kidney Injury by Promoting Autophagy.

Maomao Sun et al.

Frontiers in immunology, 12, 685523-685523 (2021-08-03)

Recent studies have shown that autophagy upregulation can attenuate sepsis-induced acute kidney injury (SAKI). The tumor suppressor p53 has emerged as an autophagy regulator in various forms of acute kidney injury (AKI). Our previous studies showed that p53 acetylation exacerbated

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全球贸易项目编号

货号	GTIN
SAB1305552-400UL	04061836266691