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SAB5200006

Sigma-Aldrich

Monoclonal Anti-CRYAA antibody produced in mouse

clone 1H3.B8, 1 mg/mL, purified immunoglobulin

别名:

Anti-Acry1, Anti-Alpha A Crystallin, Anti-CRYA1, Anti-CRYAA, Anti-Heat Shock protein beta4, Anti-HspB4

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About This Item

UNSPSC代码:
12352203
NACRES:
NA.41
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生物来源

mouse

偶联物

unconjugated

抗体形式

purified immunoglobulin

抗体产品类型

primary antibodies

克隆

1H3.B8, monoclonal

表单

buffered aqueous glycerol solution

分子量

antigen predicted mol wt 20 kDa

种属反应性

bovine, rat, mouse, human

浓度

1 mg/mL

技术

indirect ELISA: suitable
western blot: suitable

特异性

Detects α-crystallin at ~20 kDa. Does not cross-react with αB-crystallin, βL-crystallin, βH- crystallin, γ-crystallin, Hsp25, Hsp27 or Hsp47 proteins.

免疫原

Native Alpha Crystallin

特点和优势

Evaluate our antibodies with complete peace of mind. If the antibody does not perform in your application, we will issue a full credit or replacement antibody. Learn more.

外形

Solution in PBS, pH 7.2, in 50% glycerol, and 0.09% sodium azide

免责声明

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

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储存分类代码

10 - Combustible liquids

WGK

WGK 1

闪点(°F)

Not applicable

闪点(°C)

Not applicable


历史批次信息供参考:

分析证书(COA)

Lot/Batch Number

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Shiwani Sharma et al.
Molecular vision, 24, 801-817 (2019-02-05)
Pseudoexfoliation (PEX) syndrome is an age-related progressive disease of the extracellular matrix with ocular manifestations. PEX is clinically diagnosed by the presence of extracellular exfoliative deposits on the anterior surface of the ocular lens. PEX syndrome is a major risk
Andrea Schlotterer et al.
FASEB journal : official publication of the Federation of American Societies for Experimental Biology, 33(3), 4141-4153 (2018-11-30)
The aim of this study was to evaluate whether damage to the neurovascular unit in diabetes depends on reactive metabolites such as methylglyoxal (MG), and to assess its impact on retinal gene expression. Male Wistar rats were supplied with MG

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